Hidropisia fetal: análise de 80 casos

Silva, André Ricardo Araújo da; Alzegui, Júlio Cezar Laura; Costa, Maria Célia de Freitas Leite da; Tristão, Maria Aparecida; Nogueira, Susie Andries; Nascimento, Jussara Pereira do

doi:10.1590/s0100-72032005000300008

Cited by 5 publications

(3 citation statements)

References 14 publications

(29 reference statements)

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“…Hydrops was identified by prenatal ultrasound examination in 90.5% of the cases in the present study. Similar results were described in other NIHF series [Mascaretti et al, 2003;Silva et al, 2005]. In our series the registry of HF have occurred predominantly in the 2nd trimester (median: 21 weeks), more early than that reported in old series (25-32 weeks) [Mahony et al, 1984;McCoy et al, 1995;Rejjal et al, 1996;Swain et al, 1999].…”

Section: Discussionsupporting

confidence: 90%

Non‐immune hydrops fetalis: A prospective study of 53 cases

Moreno

Kanazawa

Barini

et al. 2013

American J of Med Genetics Pt A

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Non-immune hydrops fetalis (NIHF) is a symptom caused by a heterogeneous group of conditions. Diagnostic investigations may constitute a real challenge. This study aimed to evaluate prospectively and systematically a series of NIHF cases using a research protocol expanded for studying inborn errors of metabolism (IEM) during 2 years-2010 and 2011. We also reviewed the frequency of IEM among the NIHF reported in literature. A clinical or etiopathogenic diagnosis was reached in 46 (86.8%) of the 53 studied cases. The main diagnostic groups were chromosomal anomalies (28.3%), syndromic (18.9%), isolated cardiovascular anomaly (7.5%) and congenital infection (7.5%). Metabolic causes were found in 5.7%, all lysosomal storage disorders (LSD). In seven (13.2%), no diagnosis was found in part because of incomplete evaluation. The hydrops was identified prenatally in 90.5% of cases. In 5.7% a spontaneous and complete resolution of the hydrops occurred during pregnancy. Overall mortality was 75.5%. The IEM frequency in the present study (5.7%) was higher than that usually reported. We suggest performing studies directed to IEMs if the more common causes are excluded.

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Section: Discussionsupporting

confidence: 90%

Non‐immune hydrops fetalis: A prospective study of 53 cases

Moreno

Kanazawa

Barini

et al. 2013

American J of Med Genetics Pt A

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“…With the advent, in 1969, of anti-D immunoprophylaxis, non-immune fetal hydrops became more prevalent. However, there is a relative increase in the incidence of immune fetal hydrops by antibodies called minor, that is, low frequency antibodies, and therefore, in suspected cases of immune fetal hydrops, they should be investigated (Silva et al, 2005).…”

Section: Causes Of Immune Fetal Hydropsmentioning

confidence: 99%

“…Of these minor antigens, the Kell blood group system is the most prevalent, accounting for approximately 10% of cases of immune-mediated anemia (Silva et al, 2005). Still, immune fetal hydrops can arise in a context where immunological and non-immunological causes coexist or where multiple antigens are simultaneously implicated (Lee et al, 2016).…”

Section: Causes Of Immune Fetal Hydropsmentioning

confidence: 99%

Fetal hydropsia: challenges in etiologies

Dantas

Santos

Cano

et al. 2021

RSD

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Introduction: Fetal hydrops is defined as the presence of abnormal fluid collections in two or more extravascular fetal compartments and body cavities. There are about 150 different underlying causes known today potentially leading to this fetal alteration. Objective: To analyze the etiologies involved in the occurrence of cases of fetal hydrops. Methods: A systematic literature review was carried out using the MedLine, Pubmed and Scielo databases, from 2015 to 2021, using the expressions: "fetal, hydrop, etiologies." Discussion: Fetal hydrops is divided into immune and non-immune. Immune results from anemia secondary to erythroblastosis by alloimmunization, so when there is maternal exposure to fetal antigens, it generates an immune response that results in the production of antibodies. History of blood transfusions, previous births, trauma and a history of alloimmunization are characterized as risk factors. Thus, immunoprophylaxis with anti-D immunoglobulin is indicated for all RhD negative pregnant women, with RhD positive male partner, with abundant fetal maternal hemorrhage during childbirth or events with potential sensitizer in the prenatal period. Conclusion: For an effective treatment, it is essential to identify the type of fetal hydrops in the patient and then the etiology of the disease, which is quite variable in Non-Immune Fetal Hydrops.

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Prevalência de anticorpos IgG antiparvovírus B19 em gestantes durante o atendimento pré-natal e casos de hidropisia fetal não imune atribuídos ao parvovírus B19, na Cidade do Rio de Janeiro

Silva

Nogueira

Alzeguir

et al. 2006

Rev. Soc. Bras. Med. Trop.

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RESUMO ABSTRACTWith the aim of measuring the prevalence of anti-parvovirus B19 IgG antibodies during pregnancy up to 24 weeks of gestation and detecting cases of nonimmune hydrops fetalis, 249 sera from pregnant women attending a reference hospital in Rio de Janeiro City, from June 2003 to November 2004 were collected. They were followed-up until the end of pregnancy, with 17 cases of fetal hydrops detected. Four cases were caused by parvovirus B19 and two of them occurred in pregnant women living in the western zone of the city, during February 2005. Anti-parvovirus B19 IgG antibodies were found in 172 (71.6%) pregnant women (CI 95% 65.5%-77.7%); this antibody prevalence is similar to results found for others Brazilian cities. The only variable associated with previous acquisition of IgG antibodies to parvovirus B19 was number of pregnancies greater than one (p=0.02, CI 95% 0.36-0.94).

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Hidropisia fetal: análise de 80 casos

Cited by 5 publications

References 14 publications

Non‐immune hydrops fetalis: A prospective study of 53 cases

Non‐immune hydrops fetalis: A prospective study of 53 cases

Fetal hydropsia: challenges in etiologies

Prevalência de anticorpos IgG antiparvovírus B19 em gestantes durante o atendimento pré-natal e casos de hidropisia fetal não imune atribuídos ao parvovírus B19, na Cidade do Rio de Janeiro

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