Granuloma elastolítico anular de células gigantes: a propósito de un caso localizado en áreas no fotoexpuestas

Campos-Muñoz, Lucía; Diaz‐Díaz, R.M.; Quesada-Cortés, Alicia; Hernández‐Cano, Natalia; Martín-Díaz, M.A.; Regojo-Zapata, Rita M; Jiménez, Mariano Casado

doi:10.1016/s0001-7310(06)73458-5

Cited by 16 publications

(3 citation statements)

References 18 publications

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“…Generally, it is accepted that AG is a variant, the best known, of EGCG [9,10,11] and we also share this opinion. Afterwards, several authors have confirmed the existence of this entity and published similar cases in photoexposed areas, [3,8,12] but also on areas not exposed to sunlight [9,13,14].…”

Section: Discussionmentioning

confidence: 88%

“…In the particular case of AG, O'Brien's theory of actinic damage and solar elastosis is still accepted [2]. Some authors have reported cases of EGCG on non photoexposed areas or in a generalized fashion, which supports the idea that solar elastosis is not a "sine qua non" condition for the diagnosis [9,13,14].…”

Section: Discussionmentioning

confidence: 99%

“…Clinically, the lesions often appear as annular plaques with peripheral spreading and central clearing. However, cases with papular lesions exclusively that have been interpreted as very early forms have also been reported [9,13,24].…”

Section: Discussionmentioning

confidence: 99%

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Elastolytic giant cell granuloma: clinic-pathologic review of twenty cases

Gutiérrez‐González¹,

Gómez-Bernal²,

Álvarez-Pérez³

et al. 2013

Dermatology Online Journal

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Background: O'Brien described four histopathological patterns of actinic granuloma (AG). Since then, only single cases and a few series have been reported in the literature, most corresponding to cases of the giant cell type.Methods: We reviewed all the cases diagnosed as AG or elastolytic giant cell granuloma (EGCG) in our department from 1988 until 2010. The biopsies were classified into the four patterns previously described.Results: Giant cell pattern was found to be the most frequent (70% of the cases). In four cases, the biopsies showed more than one histopathologic pattern. All the lesions were located on sun-exposed areas or were related to chronic heat exposure. Diabetes mellitus was associated in 40 % of the cases. Conclusions:The giant cell pattern of EGCG is the most frequent. Some cases may share histopathologic features of more than one variant and thus, we consider they may be categorized as mixed patterns. Diabetes mellitus is the most common associated disease and should always be ruled out.

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Section: Discussionmentioning

confidence: 88%

Section: Discussionmentioning

confidence: 99%

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Elastolytic giant cell granuloma: clinic-pathologic review of twenty cases

Gutiérrez‐González¹,

Gómez-Bernal²,

Álvarez-Pérez³

et al. 2013

Dermatology Online Journal

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Acquired Disorders of Dermal Connective Tissue

Lovell

2016

Rook's Textbook of Dermatology, Ninth Edition

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The subject matter of this chapter comprises the major acquired disorders that affect mainly collagen and elastic tissue. The connective tissue of normal skin is discussed elsewhere. Heritable disorders of connective tissue, such as Ehlers–Danlos syndrome, are covered in another chapter, as are disorders of ground substance, such as the mucinoses and mucopolysaccharidoses. In addition, many disorders, including developmental defects, neoplasms and other reactive or scarring conditions, may involve the connective tissues. Although it can be convenient to separate conditions into disorders affecting collagen or elastic tissue, the distinction is somewhat artificial, as changes in these proteins are generally preceded by abnormal metabolism of other matrix components, such as fibronectin or periostin.

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Acquired Disorders of Dermal Connective Tissue

Fahy,

Lovell

2024

Rook's Textbook of Dermatology

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This chapter reviews acquired changes in dermal connective tissue during normal ageing and in disease processes, including photodamage. Corticosteroids are a potent cause of dermal atrophy, which may also be congenital or induced by inflammatory disorders related to morphoea. Enzymatic degradation of elastic tissue results in localised or generalised skin laxity, which resembles genetic disorders such as pseudoxanthoma elasticum and cutis laxa. In contrast, some disorders are characterised by excessive deposition of collagen or elastic tissue. Fibrosis is associated with contracture, which causes deformity and restricted movement as in palmoplantar fibromatosis. Several chemical environmental triggers induce diffuse fibrosis resembling systemic sclerosis. Keloids represent an abnormal scarring response to trauma. Finally, dermal connective tissue components may be extruded via the epidermis in perforating disorders such as elastosis perforans serpiginosa.

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Granuloma elastolítico anular de células gigantes: a propósito de un caso localizado en áreas no fotoexpuestas

Cited by 16 publications

References 18 publications

Elastolytic giant cell granuloma: clinic-pathologic review of twenty cases

Elastolytic giant cell granuloma: clinic-pathologic review of twenty cases

Acquired Disorders of Dermal Connective Tissue

Acquired Disorders of Dermal Connective Tissue

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