Giant Inflammatory Polyposis Causing Partial Obstruction and Pain in “Healed” Ulcerative Colitis in an Adolescent

Adelson, JW; Jp, deChadarévian; Em, Azouz; Fm, Guttman

doi:10.1097/00005176-198801000-00025

Cited by 10 publications

(7 citation statements)

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“…Most published reports of inflammatory polyposis have described the occurrence of this syndrome in patients with known pre‐existing inflammatory bowel disease. Approximately 20% of adult patients with long‐standing ulcerative colitis develop inflammatory polyposis 82 . The incidence of this condition in Crohn's disease is not known, but in one series nearly one‐third of patients with Crohn's disease also had inflammatory polyposis 81 .…”

Section: Inflammatory Polyposismentioning

confidence: 99%

“…Inflammatory polyposis can develop in patients within a short period of time after the onset of clinically recognized inflammation, and pseudopolyps may be seen during the first acute attack of inflammatory bowel disease 83 . Inflammatory polyps are seen in patients with active or quiescent inflammatory bowel disease 82 , . 84 Patients with inflammatory bowel disease complicated by dilation and megacolon are also more likely to have inflammatory polyposis 83 .…”

Section: Inflammatory Polyposismentioning

confidence: 99%

“…Besides the well‐recognized symptoms and complications that are usually associated with inflammatory bowel disease, the presence of inflammatory polyposis may be associated with unique complications. Colonic obstruction has been caused by inflammatory polyposis in patients with ulcerative colitis, even in the absence of acute inflammation 82 , . 85 Protein‐losing enteropathy has also occurred in this setting 80 , .…”

Section: Inflammatory Polyposismentioning

confidence: 99%

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The non‐inherited gastrointestinal polyposis syndromes

Ward

Wolfsen

2002

Aliment Pharmacol Ther

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The non‐inherited gastrointestinal polyposis syndromes represent a group of rare disorders characterized by the presence of multiple, non‐adenomatous polyps on the gastrointestinal mucosa occurring in unrelated patients. We present here a review of the clinical and histo‐ pathological aspects of the syndromes to include the Cronkhite–Canada syndrome, hyperplastic polyposis and lipomatous polyposis. While infrequently encoun‐ tered, these diseases can have devastating clinical effects that may be aggravated by delays in diagnosis and treatment. Prompt accurate diagnosis and treatment of these uncommon disorders depend on a sound working knowledge of the distinct clinical and pathological features described herein.

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Section: Inflammatory Polyposismentioning

confidence: 99%

Section: Inflammatory Polyposismentioning

confidence: 99%

Section: Inflammatory Polyposismentioning

confidence: 99%

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The non‐inherited gastrointestinal polyposis syndromes

Ward

Wolfsen

2002

Aliment Pharmacol Ther

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“… 10 More than 70 cases of giant pseudopolyposis associated with inflammatory bowel disease have been reported. 2,4–7,10–42 The presence of pseudopolyps per se does not reflect the severity of the underlying inflammatory bowel disease and may be found in both active and quiescent phases of ulcerative colitis. 2 The reported duration of ulcerative colitis and Crohn’s colitis, in whom pseudopolyposis is found, ranges, respectively, from 1 month to 20 years (median, 5 years) 2,4,10–34 and 3 months to 37 years (median, 6 years).…”

Section: Introductionmentioning

confidence: 99%

Giant Pseudopolyposis in Inflammatory Bowel Disease

Ooi

Tjandra

Pedersen

et al. 2000

Aust. N.Z. J. Surg.

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“…Inflammatory polyps may produce symptoms of pain and obstruction [246] and may be pedunculated or filiform. They may occur in settings of minimal inflammation, such as ulcerative colitis in remission, or in a familial setting [247].…”

Section: Inflammatory Polypsmentioning

confidence: 99%

Neoplastic and Nonneoplastic Polyps of the Colon and Rectum

Macrae

Young

2008

Textbook of Gastroenterology

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Epidemiology PrevalenceInformation on the prevalence of polyps comes from autopsy studies and from colonoscopic surveys, which are of a much smaller scale. Autopsy studies generally show a higher prevalence of polyps and adenomas than endoscopic studies. Prevalence rates vary considerably between gender (males more common), ethnic groups, and countries, and reflect the rates of colorectal cancer in those population groups (see Chapter 63). In the United States, autopsy studies suggest an overall polyp prevalence of up to 50% [6,7], but colonoscopic surveys suggest no more than 40% [8][9][10][11][12][13][14][15]. In autopsy studies, fixation of the colon and careful use of a magnifying glass allow detection of small polyps that are more readily missed during colonoscopy [16][17][18][19][20].More than 90% of polyps detected at colonoscopy are epithelial in origin, that is, adenomas or hyperplastic polyps, and 75% of these are adenomas [8][9][10][11][12][13]. In the case of diminutive epithelial polyps (less than 5 mm), about half are hyperplastic [14,15].Age is the major determinant of adenoma prevalence [6,7]. Size at detection also increases with age [20]. Colorectal adenomas are uncommon in patients younger than 30 years of age. Anatomical distributionStudies show that colorectal adenomas are more common in the distal colon and rectum, similar to the distribution of colorectal cancer (see Chapter 63). Distribution, however, relates to size. Small adenomas are more uniformly distributed throughout the entire colon, whereas large adenomas (greater than 1 cm) show a distal predominance [7,8,21,22].

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Giant Inflammatory Polyposis Causing Partial Obstruction and Pain in “Healed” Ulcerative Colitis in an Adolescent

Cited by 10 publications

References 0 publications

The non‐inherited gastrointestinal polyposis syndromes

The non‐inherited gastrointestinal polyposis syndromes

Giant Pseudopolyposis in Inflammatory Bowel Disease

Neoplastic and Nonneoplastic Polyps of the Colon and Rectum

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