Genomic structure and embryonic expression of estrogen receptor beta a (ERβa) in zebrafish ( Danio rerio )

Early regulation of brain aromatase (cyp19a1b) by estrogen receptors during zebrafish development

“…The present study shows that significant amounts of esr2a mRNA are maternally inherited, as already shown by real-time RT-PCR or RNAse protection (Lassiter et al, 2002;TingaudSequeira et al, 2004;Bardet et al, 2005). The amounts of maternally in- herited esr1 and esr2b mRNA are in contrast much lower.…”

Differentiation of murine embryonic stem cells induces progesterone receptor gene expression

“…In the developing mouse embryo, ERα, ERβ, and PR are expressed at the blastocyst stage and thereafter [16][17][18][19][20][21][22][23]. The expression of these receptors in mES cells may not directly reflect the same timing as in development, but their expression in mES cells allows us to use it as a model system of in vitro differentiation.…”

“…Other members of the nuclear receptor family, estrogen receptor alpha (ERα), estrogen receptor beta (ERβ), and progesterone receptor (PR) are present in the early developing embryo of mice and other species [16][17][18][19][20][21][22][23]. In particular, ERα is present in the oocyte, shows variable expression through the morula stage, followed by increased expression in the inner cell mass of the blastocyst.…”

Effects of steroid hormones on reproduction- and detoxification-related gene expression in adult male mosquitofish, Gambusia affinis

“…It has been previously shown that the presence of an estrogen-responsive element (ERE) in the promoter of ERα in the rainbow trout may be a major cis-acting element involved in the regulation of the gene by E2 (Le Dréan et al, 1995). In addition, an imperfect ERE in the promoter of ERα has been demonstrated in the zebrafish along with ERE half sites with no consensus EREs in the promoter of ERβ (Lassiter et al, 2002;Menuet et al, 2004). It is still unclear whether the presence of different EREs in the promoters of different ER genes instructs their response to E2.…”