Gastroparesis

Hasler, William L.

doi:10.1097/mog.0b013e328358d619

Cited by 37 publications

(22 citation statements)

References 49 publications

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“…There is no evidence from the cases in this present report to conclusively prove this theory. Although gastric motility disorders are poorly described in horses, the histopathology performed in Case 1 did not identify features to support a generalised motility disorder of the stomach, as has been reported in man (Hasler ).…”

Section: Discussionmentioning

confidence: 70%

Gastric impaction associated with displacement and volvulus of the large colon in seven mature horses

McGovern¹,

Suthers²,

James³

et al. 2015

Equine Veterinary Education

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Summary Gastric impaction associated with large colon volvulus (LCV) was identified in seven horses. Right dorsal displacement of the large colon and suspected nephrosplenic entrapment was identified in 2 of the 7 horses as well as LCV with concurrent gastric impaction. All horses underwent surgery for LCV and none survived. Five horses died or were subjected to euthanasia intraoperatively or in recovery. One horse was subjected to euthanasia post operatively due to persistent gastric reflux, following resolution of the gastric impaction. One horse was subjected to euthanasia post operatively due to a suspected gastric rupture, which was confirmed on post mortem examination. It is hypothesised that a large mass in the cranial abdomen, such as a gastric impaction may disrupt the normal anatomical large colon alignment or may cause colonic motility or microbiota alterations, and thus increase the risk of large colon displacement and volvulus.

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Section: Discussionmentioning

confidence: 70%

Gastric impaction associated with displacement and volvulus of the large colon in seven mature horses

McGovern¹,

Suthers²,

James³

et al. 2015

Equine Veterinary Education

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“…The etiology of iron deficiency is multifactorial, including dietary limitations, decreased gastric acidity and SIBO. As ferrous iron is required for the absorption of dietary iron, reduced gastric acidity in patients with GP and the treatment with PPIs results in increased concentrations of non‐absorbable ferric ion . Furthermore, patients receiving jejunal EN should be monitored for iron deficiency as the duodenum (the primary site of iron absorption) is bypassed.…”

Section: Malnutritionmentioning

confidence: 99%

Management of gastroparesis‐associated malnutrition

Bharadwaj

Meka

Tandon

et al. 2016

J of Digest Diseases

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Gastroparesis (GP) is a chronic debilitating dysmotility characterized by unrelenting nausea, vomiting, bloating, early satiety, postprandial fullness and abdominal pain. Patients with GP experience other associated conditions, including gastroesophageal reflux disease, gastric bezoars and small bowel bacterial overgrowth. Furthermore, GP is associated with poor quality of life, increased emergency room visits, hospitalizations and subsequent increased healthcare costs. Currently, the managements of GP consist of glycemic control, antiemetics, prokinetics and the use of gastric electrical stimulation. However, most GP patients are at risk for significant nutritional abnormalities. As such, it is essential to screen and diagnose malnutrition in these patients. Poor oral intake in such patients could be supplemented by enteral tube feeding. Parenteral nutrition, although a last resort, is associated with a number of complications and should be used only for the short term. In summary, a systematic approach including initial nutritional screening, diet recommendations, medical therapy, nutritional re-evaluation and enteral and parental nutrition should be considered in complex GP patients.

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“…2 Our patient responded well to this approach, but gastrojejunostomy tubes and gastric pacing devices may be helpful in recalcitrant cases. 1,2 …”

mentioning

confidence: 93%

“…Gastric motility studies are the diagnostic gold standard. 2 Diabetic gastroparesis can be asymptomatic or it may manifest with postprandial fullness, nonspecific epigastric discomfort and weight loss. The pathophysiology is believed to involve some combination of autonomic neuropathy and dysfunction of Cajal pacemaker cells, although hyperglycemia can also delay gastric emptying.…”

mentioning

confidence: 99%

“…The pathophysiology is believed to involve some combination of autonomic neuropathy and dysfunction of Cajal pacemaker cells, although hyperglycemia can also delay gastric emptying. 1,2 Conservative treatment consists of dietary changes, blood glucose management and the judicious use of promotility agents. 2 Our patient responded well to this approach, but gastrojejunostomy tubes and gastric pacing devices may be helpful in recalcitrant cases.…”

mentioning

confidence: 99%

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