Fundamental frequency, language processing, and linguistic structure in Wernicke's aphasia

Some Acoustic Aspects of Fundamental Frequency of CVC Utterances in Aphasia

“…In studying the distribution of the F0 over sentence-sized units, several studies have attributed the deficit that they have found to a problem in syntactic planning [Danly and Shapiro, 1982;Danly et al, 1983]. But it remains to be determined if it is only when sentence-or phrase-sized units are investigated, which necessitate a higherlevel planning, that the deficit in F0 produc tion occurs.…”

“…In simplistic terms, anterior apha sies know what they want to say, but cannot produce it very well, while posterior apha sies produce quite well what they do not know how to say. While this result is some what surprising there is some evidence of a problem for posterior aphasies in F0 pro duction at higher levels of language pro cessing (the sentential level) [Coopérét al., 1979;Danly et al, 1983]. Perhaps part of the reason that posterior aphasies are not noted to have a speech production problem is that such studies have largely concen trated on the supralaryngeal aspects or tim ing relations in speech production [Blum stein et al, 1977[Blum stein et al, , 1980Itoh et al, 1979Itoh et al, , 1980Keller, 1975;Ryalls, 1981] and have not investigated phonation itself.…”

Neural substrates of linguistic prosody: Evidence from syntactic disambiguation in the productions of brain-damaged patients

“…However, investigations of the control of F0 in brain-damaged speakers have yielded mixed results, sometimes demonstrating deficits on the part of LHD speakers (e.g., Cooper et al, 1984;Danly & Shapiro, 1982;Danly et al, 1983;Ryalls, 1982) and sometimes showing deficits for RHD speakers (e.g., Behrens, 1989;Bradvik et al, 1991;Bryan, 1989;Shapiro & Danly, 1985;Weintraub et al, 1981). (See also Mayer and colleagues, 1999; for some interesting fMRI findings with respect to prosody production.…”

“…For example, looking only at linguistic prosody, numerous investigators have reported impairments in the control of temporal parameters of speech prosody in lefthemisphere-damaged (LHD) individuals (e.g., Baum et al, 2001;Danly & Shapiro, 1982;Danly et al, 1983;Gandour et al, 1993;2000;Schirmer et al, 2001), but relatively spared control of timing in individuals with right hemisphere damage (RHD) (Baum & Boyczuk, 1999;Gandour et al, 1994;Schirmer et al, 2001;Walker et al, 2004). However, investigations of the control of F0 in brain-damaged speakers have yielded mixed results, sometimes demonstrating deficits on the part of LHD speakers (e.g., Cooper et al, 1984;Danly & Shapiro, 1982;Danly et al, 1983;Ryalls, 1982) and sometimes showing deficits for RHD speakers (e.g., Behrens, 1989;Bradvik et al, 1991;Bryan, 1989;Shapiro & Danly, 1985;Weintraub et al, 1981).…”

Aquisição, afasia e a hierarquia prosódica

“…Neste tipo de afasia, acredita-se, a altura, o acento e a duração encontram-se bem preservados, com exceção de algumas características que podem distinguir a fala de alguns sujeitos assim afásicos da de sujeitos não cérebro-lesados. A divisão do enunciado em unidades menores é mais frequente do que em sujeitos normais ( (Danly, Cooper & Shapiro 1983), além do que tais sujeitos afásicos exibem uso mais frequente de "F o resetting": iniciar um novo contorno entonacional ou uma nova fronteira prosódica depois de uma fronteira sintática maior. Além disso, há mais curvas ascendentes de "continuação".…”