Frosted Branch Angiitis With Ocular Toxoplasmosis

Je, Ysasaga; Davis, Janet L.

doi:10.1001/archfami.9.10.962

Cited by 3 publications

(3 citation statements)

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“…41 The cases listed in Table 1 and referred to above appear, with limited variation, to adhere to Ito's original description of a primary idiopathic FBA. However, other cases of FBA, often localised, have been described, which appear either to be secondary to intraocular infection by CMV, [51][52][53][54][55][56][57][58] HSV (confirmed by aqueous PCR), 59,60 herpesvirus hominis, 61 toxoplasmosis 62 or Fusarium dimerium, 63 or in association with systemic lupus erythematosus, 64 Crohn's disease, 65 lymphoma, 66 leukaemia 67 and Behçet's disease (our own unpublished case, Figure 4). In summary, Frosted branch angiitis in typical form, unassociated with preexisting intraocular inflammation, is likely to be a primary immunological process in response to a number of provoking antigens.…”

Section: Discussion and Review Of The Literaturementioning

confidence: 89%

Frosted branch angiitis: a review

Walker

Iguchi

Jones

2004

Eye

107

151

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The purpose of this study is to present the first report of a case of primary frosted branch angiitis from the UK and to review the characteristics of this rare disease. Primary frosted branch angiitis causes characteristic florid translucent retinal perivascular sheathing of both arterioles and venules in association with variable uveitis, retinal oedema and visual loss, normally with good recovery. A total of 57 cases have been reported in the world literature. Atypical, typically focal frosted branch angiitis may also occur secondary to other causes of intraocular inflammation, especially cytomegalovirus retinitis. Primary frosted branch angiitis has a characteristic presentation but a variable course, typically affecting children or young adults. The disease is likely to represent a common immune pathway in response to multiple infective agents. The optimal treatment is unclear.

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Section: Discussion and Review Of The Literaturementioning

confidence: 89%

Frosted branch angiitis: a review

Walker

Iguchi

Jones

2004

Eye

107

151

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“…Although periarteritis related to toxoplasmosis is common, and toxoplasmic retinal vasculitis without focal necrotizing retinochoroiditis is known, there have also been a few reports of frosted branch angiitis associated with toxoplasmosis (Ysasaga & Davis 1999).…”

Section: Discussionmentioning

confidence: 99%

Recurrent secondary frosted branch angiitis after toxoplasmosis vasculitis

Huh

Kim

2005

Acta Ophthalmologica Scandinavica

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ABSTRACT.Purpose: To describe a case of recurrent frosted branch angiitis after treatment of ocular toxoplasmosis. Methods: In a 6-year-old boy, we found perivascular, creamy, patchy, retinal sheathing in both eyes without any focal necrotizing retinochoroiditis or scarring. IgM antibodies for toxoplasma gondii were also found. The patient was treated with antitoxoplasmosis medication and a systemic steroid. Results: Several years after treatment of the toxoplasmosis, frosted branch angiitis occurred twice without any retinal scarring or serological evidence of toxoplasmosis. After systemic steroid therapy, the angiitis improved without further complications. Conclusions: Toxoplasmic retinal vasculitis should be considered as a cause of frosted branch angiitis.

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“…The identification of underlying causes has important implications for therapy. Reported conditions that can present with secondary FBA include herpes simplex virus retinitis, varicella zoster virus retinitis, cytomegalovirus retinitis, toxoplasmosis, tuberculosis, familial Mediterranean fever, antiphospholipid antibody syndrome, systemic lupus erythematosus, syphilis, HIV infection, Behçet's disease, Hodgkin lymphoma (HL), and direct ocular invasion by malignant cells …”

Section: Introductionmentioning

confidence: 99%