Fetal renal dysplasia: sonographic evaluation.

Mahony, B S; Filly, Roy A.; Callen, P W; Hricak, H; Golbus, M S; Harrison, M R

doi:10.1148/radiology.152.1.6729104

Cited by 109 publications

(24 citation statements)

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“…3 The presence of dysplasia implies irreversible kidney damage. 9 Both bladder outlet obstruction and renal dysplasia contribute to oligohydramnios which has been associated with impaired pulmonary development and decreased survival. 2…”

Section: Discussionmentioning

confidence: 99%

Posterior urethral obstruction. Prenatal sonographic findings and clinical outcome in fourteen cases.

Hayden

Russ

Pretorius

et al. 1988

Journal of Ultrasound in Medicine

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Fourteen cases of fetal urethral obstruction were reviewed retrospectively. The purpose of this study was to emphasize the following: 1) prenatal sonographic find· ings: 2) clinical outcome: and 3) associated congenital anomalies. Decreased amniotic fluid volume complicated 12 pregnancies (86%). A dilated posterior urethra was identified in nine fetuses (64%) and an enlarged bladder in 13 (93%). Evaluation of the fetal kidneys re· P osterior urethral abnormalities are a common cause of bladder outlet obstruction and bilateral hydronephrosis. u Congenital urinary tract obstruction can result in renal dysplasia and impaired pulmonary development. 3 -6 Evidence suggests that in utero decompression of the urinary tract reverses the effects of obstruction. The efficacy of this procedure, however, remains questionable without a complete understanding of the natural history of fetal obstructive uropathy. The decision to intervene is consequently dependent on the presence of associated congenital anomalies. s:; Details concerning the antenatal evaluation of posterior urethral obstruction have been discussed in a lim· ited number of previous reports.U 371, 1988) emphasize its natural history and to stress its association with other congenital anomalies. MATERIALS AND METHODSFourteen fetuses with urethral obstruction were evaluated in our ultrasound department over a 5-year period from 1982 to 1986. Data were compiled by retrospective review of available sonograms, ultrasound reports, and medical records. If more than one ultrasound examination had been obtained, findings from the first study were recorded. Follow-up sonograms were not included, since our intent was to review findings at initial presentation rather than to evaluate longitudinal changes that might occur in utero. For each case, the following sonographic parameters were assessed: 1) gestational age, 2) amniotic fluid volume, 3) fetal gender, 4) urethral dilatation, 5) bladder size, 6) bladder wall hypertrophy, 7) hydronephrosis, 8) renal echogenicity, 9) renal cysts, and 10) fetal ascites. Additional data collected included pregnancy outcome, clinical course, and associated congenital abnormalities. Autopsy reports were available in nine cases. In four of the fetuses, not every sonographic pa-

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Section: Discussionmentioning

confidence: 99%

Posterior urethral obstruction. Prenatal sonographic findings and clinical outcome in fourteen cases.

Hayden

Russ

Pretorius

et al. 1988

Journal of Ultrasound in Medicine

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show abstract

“…Since collecting system dilatation may suggest physiologic compromise, 6 recognition of these findings is important. as obstetric and perinatal management may be altered..…”

Section: Discussionmentioning

confidence: 99%

Sonographic diagnosis of bilateral fetal renal duplication with ectopic ureteroceles.

Schoenecker

Cyr

Mack

et al. 1985

Journal of Ultrasound in Medicine

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“…Group I : classical multicystic dysplasia resulting from ureteral atresia at pelviureteric junction, Group II : focal and segmental cystic renal dysplasia usually caused by obstruction related to ectopic uretrocele with ureteral duplication, Group III : cystic renal dysplasia associated with lower urinary tract obstruction e.g. posterior urethral valves and Group IV : heredofamilial renal dysplasia without any obstructive uropathy [6].…”

Section: Discussionmentioning

confidence: 99%