“…The GI is secondary at the migration of a gallstone to the bowel through a fistula [7] , [9] , [10] , [13] , formed due a chronic inflammation in the gallbladder and the local pressure by the gallstone against, most frequently, the small bowel [8] , [9] , [13] . The cholecytoduodenal fistula is the most common, in 85% of the cases, followed by cholecystogastric, cholecystojejunal and cholecystocolonic [1] , [3] , [5] , [12] , [13] , [14] . There are other rarest cases, such a fistula originated directly from the biliary tract, with a higher rate of cholangitis [7] , the passage of gallstones through the ampulla of Vater followed by in-situ growth or inadvertent iatrogenic migration of gallstone during manipulation of gallbladder or ducts [1] , [3] , [13] , [14] .…”