Family history of periodontal disease and prevalence of smoking status among adult periodontitis patients: a cross‐sectional study

Abstract

Within the investigated population familial aggregation, smoking status, age and gender are factors that were related to extent and severity of adult periodontitis.

Prevalence of diabetes among patients diagnosed with periodontitis: A retrospective cross‐sectional study

“…The prevalence of smoking was 34%; 37% were never smokers and 29% reported to be past smokers. The results also showed a significantly higher predictive value for smokers to belong to a periodontitis group with a greater extent of periodontal destruction …”

“…A wealth of epidemiological, clinical and in vitro studies has emerged that have provided irrefutable evidence that smoking negatively impacts periodontal health and proposes mechanisms by which this may occur. Based on the database of the present study, the prevalence of smokers and the impact on extent and severity of periodontitis among this population has been reported . The prevalence of smoking was 34%; 37% were never smokers and 29% reported to be past smokers.…”

“…Although this approach is not widely used therefore, preventing comparisons with other studies, the Van der Velden classifications system can be used for purposes such as estimates of treatment needs, identification of risk factors and disease activity . This paper and a recent publication from our group present an initial report on the differentiation of extent and severity among a population of periodontitis patients in relation to the prevalence of putative risk factors such as age, gender, family history of periodontitis and smoking.…”

Analysis of salivary phenotypes of generalized aggressive and chronic periodontitis through nuclear magnetic resonance‐based metabolomics

“…The latter finding is in agreement with an increasing body of evidence and confirms that it is almost impossible to use the term AgP as long as there is no proper way to diagnose the disease . Indeed, the discrimination between CP and AgP is not supported by sufficiently distinct histologic, microbiologic, immunologic, or genetic foundations . Moreover, microbiome exhibits conserved metabolic and virulence gene expression profiles despite the interindividual differences in the disease phenotype .…”

“…32 Indeed, the discrimination between CP and AgP is not supported by sufficiently distinct histologic, microbiologic, immunologic, or genetic foundations. [33][34][35][36][37] Moreover, microbiome exhibits conserved metabolic and virulence gene expression profiles despite the interindividual differences in the disease phenotype. 38 This may suggest that what distinguishes AgP from CP are dissimilarities in the immune-inflammatory host response 39 or, as advocated by Van der Velden, 32 a difference in the degree of bacterial invasiveness.…”