“… 45 Several studies have reported that levels of pro-inflammatory cytokines and chemokines, such as IL-1, IL-4, IL-6, IL-8, IL-12, TGF-β, TNF-α, prostaglandin (PG), growth factor, MCP-1, and regulated upon activation, normal T cell expressed and secreted (RANTES), are higher in the PF of women affected by endometriosis compared to disease-free participants. 83–85 These inflammatory mediators may participate in the establishment and advancement of endometriosis, and they are secreted by activated macrophages, lymphocytes, endometriotic lesions, and peritoneal mesothelial cells, thereby promoting the viability, proliferation, adhesion, invasiveness, and angiogenesis of endometrial cells. 86 …”