Endoleak and Endotension Following Open Abdominal Aortic Aneurysm Repair: A Report of Two Cases

Endotension following endovascular aneurysm repair

“…18 According to the report by Arya and colleagues, seroma, which is probably caused by fibroblast ingestion, ultrafiltration, and/or bacterial biofilm infection, cannot be avoided only by aspiration, fenestration, or resection. 19 As mentioned above, we first demonstrated increased intrasac pressure in the absence of endoleak in dogs, for which EVAR was performed using PTFE or Dacron (Coknit) graft with high porosity. Despite an increase in intrasac pressure with Dacron graft, endoleak was not detected by angiography and contrast CT. 2 Although graft material has been postulated to be responsible for the local production and transmigration of serous fluid and sac hygroma after EVAR and open AAA surgery, the relationship between the material of graft or prosthesis and the incidence of aneurysm sac enlargement remains unclear.…”

“…There are several mechanisms by which pressure can be transmitted to the aneurysm sac. [6][7][8][9][10][11][12][13][14][15][16][17][18][19] The transmission of pressure from the aorta to the aneurysm sac can occur via thrombus layered between the aortic wall and the stent-graft (Table 1). The pressure may also be transmitted through the graft wall.…”

Frequency, risk factors, and management of perigraft seroma after open abdominal aortic aneurysm repair

“…[8][9][10][11][12] Only 2 cases of a PGS after Dacron graft aortic repair have been reported. 5,14 In the current study, all 20 patients with seromas had PTFE reconstructions. This did not reach significance on univariate analysis due probably to type II statistical error.…”

Successful endovascular management of endoleak-like phenomenon following open abdominal aortic aneurysm repair

“…Type II endoleaks are the most common endoleaks encountered in clinical practice, accounting for approximately 40% of all endoleaks [9]. They occur when there is retrograde flow of blood into the aneurysm sac via an excluded aortic branch, most commonly the inferior mesenteric artery or a lumbar artery.…”

“…They occur when there is retrograde flow of blood into the aneurysm sac via an excluded aortic branch, most commonly the inferior mesenteric artery or a lumbar artery. Many type II endoleaks close spontaneously over time [9]. However, data suggest that patients with a persistent type II endoleak (>6 months) should be considered for more frequent follow-up and/or a more aggressive approach to reintervention [10].…”