Endocrine and metabolic differences among phenotypic expressions of polycystic ovary syndrome according to the 2003 Rotterdam consensus criteria

Diabetes risk score in the diagnostic categories of polycystic ovary syndrome

“…Although elevated CVD and DM2 risk factors are previously reported for NIH compared with non-NIH PCOS (27)(28)(29)(30) or non-NIH PCOS compared with controls (29,31), this is less marked for adiposity or abdominal adiposity-matched populations (reviewed by Moran and Teede [16]). The BMI, waist circumference, or waist-hip ratio-matched NIH and non-NIH PCOS (26,32) or non-NIH PCOS and controls (32)(33)(34) have similar DM2 and CVD risk profiles including fasting or post-OGTT glucose, insulin resistance, and lipids. This is not consistently observed and insulin resistance or glucose intolerance was reported for NIH compared with ovulatory (31) or nonhyperandrogenic (30) non-NIH PCOS or for ovulatory non-NIH PCOS compared with controls (35), despite comparison of obese subsets only (30) or similar weight and waist-hip ratio (31,35).…”

Relationship Between Health-Promoting Lifestyle and Quality of Life in Women With Polycystic Ovarian Syndrome

“…According to the data of World Health Organization (WHO) and World Bank, in 2002 wars had been among the first ten reasons which killed the most and caused disabilities. Civil losses are at the rate of 90% within all losses (1). War has many negative effects on human health.…”

Ovarian function and obesity—Interrelationship, impact on women's reproductive lifespan and treatment options

“…Nevertheless, the underlying mechanisms involved in IR in women with PCOS remain somewhat elusive (Kaufman et al, 2008). In the setting of IR and its subsequent hyperinsulinaemia, women with PCOS have increased risk of long-term sequelae, including increased risk of impaired glucose tolerance (IGT), T2DM nd cardiovascular disease (CVD), all exacerbated by coexistent obesity (Boudreaux et al, 2006).…”