Eleven Adolescent Girls with Severe Anorexia

Jd, Gryboski; Katz, Jeffrey N.; Mh, Sangree; Herskovic, Teodoro

doi:10.1177/000992286800701111

Cited by 27 publications

(7 citation statements)

References 14 publications

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“…Similarly, it may be difficult to diagnose organic disease in the presence of psychiatric symptoms, which may even be related to failure of the medical attendants to diagnose and treat the underlying organic disease. Though Crohn's disease is known to mimic anorexia nervosa (Silverman, 1966;Gryboski, Katz, Sangree, and Herskovic, 1968), this occurred only once in the present series, possibly because there were not many adolescent girls included. Another 14year-old girl initially presented with a fever which remitted after steroid therapy, but two years later she had a relapse of the disease which was clinically indistinguishable from anorexia nervosa.…”

Section: Discussionmentioning

confidence: 70%

Diagnosis of Crohn's disease. A continuing source of error.

Dyer¹,

Dawson²

1970

BMJ

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Section: Discussionmentioning

confidence: 70%

Diagnosis of Crohn's disease. A continuing source of error.

Dyer¹,

Dawson²

1970

BMJ

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“…It would seem that if IgA cannot protect the gut and IgM is inadequate, then the cellular components proliferate in an attempt to compensate and can result in lymphoid nodular hyperplasia, and in half the subjects splenomegaly also occurs. Fresh frozen whole plasma can ameliorate symptoms (Gryboski, Self, Clemett, and Herskovic, 1968) although anaphylactic reactions from antibodies to IgA must be avoided. Lymphoid nodular hyperplasia in the large gut can be an extension of the above, but is much more often of little clinical significance (Franken, 1970).…”

Section: Nodular Lymphoid Hyperplasiamentioning

confidence: 99%

Immunological disturbances in the pathogenesis of malabsorption

Hobbs¹

1971

Journal of Clinical Pathology

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“…It was present in the large intestine in five of the patients. A 13-yr-old boy with nodular lymphoid hyperplasia of the small intestine was reported by Gryboski, Self, Clemett, and Herskovic (1968). Serum IgG and IgM were normal but the IgA was low.…”

Section: Discussionmentioning

confidence: 99%

Nodular lymphoid hyperplasia with hypogammaglobulinaemia

Ajdukiewicz¹,

Youngs²,

Bouchier³

1972

Gut

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SUMMARY A patient with nodular lymphoid hyperplasia, who manifested defects in cellular immunity, is presented. The clinical course is discussed and a review included of the 23 patients thus far reported with nodular lymphoid hyperplasia and hypogammaglobulinaemia.Acquired idiopathic hypogammaglobulinaemia can be associated with a sprue-like syndrome in about 20% of patients (Gitlin, Gross, and Janeway, 1959). Hermans, Huizenga, Hoffman, Brown, and Markowitz (1966) described the association of hypogammaglobulinaemia with nodular lymphoid hyperplasia of the small intestine, recurrent upper respiratory infections, the malabsorption syndrome with steatorrhoea, and infestation with Giardi lamblia. Nodular lymphoid hyperplasia is relatively rare and is not found in the vast majority of patients with acquired idiopathic hypogammaglobulinaemia. Our purpose is to report a patient with this disorder and to review the literature relating to this subject. Case HistoryThe patient, a male aged 62, presented in November 1969 complaining of loss of appetite and vomiting for two weeks and weight loss of 20 kg over one month. His previous health had been good apart from the symptoms of chronic bronchitis for 30 years. Examination showed evident weight loss and pallor. Haemoglobin was 9.5 g per 100 ml with macrocytosis of the peripheral blood film. Megaloblasts were present in the sternal marrow biopsy. An augmented histamine test revealed achlorhydria. After a standard Schilling test no radioactive vitamin B12 was excreted in the urine and the addition of intrinsic factor improved the 24-hour urinary excretion to only 7 % of the administered dose (normal > 12 %). Total serum protein was 4.6 g per 100 ml (albumin 2-9 g, total globulin 1-7 g). A barium meal showed an atrophic mucosal pattern and poor peristalsis suggestive of linitis plastica.After transfusion with 4 pints of blood a laparotomy revealed no abnormality except for a few enlarged mesenteric nodes, one of which was

show abstract

Eleven Adolescent Girls with Severe Anorexia

Cited by 27 publications

References 14 publications

Diagnosis of Crohn's disease. A continuing source of error.

Diagnosis of Crohn's disease. A continuing source of error.

Immunological disturbances in the pathogenesis of malabsorption

Nodular lymphoid hyperplasia with hypogammaglobulinaemia

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