Electroencephalographic Changes in Acute Cerebral Anoxia From Cardiac or Respiratory Arrest

Hockaday, Judith M.; Potts, Frances; Epstein, Eve; Bonazzi, Anthony; Schwab, Robert S.; Brechner, Verne L.

doi:10.1097/00132586-196606000-00007

Cited by 20 publications

(23 citation statements)

References 0 publications

Supporting

Mentioning

Contrasting

Unclassified

Order By: Relevance

“…Although attempts were made to limit the effect of sedatives, there remains a possibility that residual medications still had a confounding effect on some examinations. Our dichotomized definition of poor prognostic indicators on EEG was operational and other patterns previously reported to be associated with poor outcome (ie, alpha coma, periodic epileptiform discharges) could be considered 24–27. However, such patterns were seen very infrequently in our patients.…”

Section: Discussionmentioning

confidence: 90%

Predictors of neurologic outcome in hypothermia after cardiac arrest

Fugate

Wijdicks²,

Mandrekar³

et al. 2010

Annals of Neurology

306

206

View full text Add to dashboard Cite

Clinical examination (brainstem reflexes, motor response, and presence of myoclonus) at Day 3 after cardiac arrest remains an accurate predictor of outcome after therapeutic hypothermia. Sedative medications in both hypothermic and nonhypothermic patients may confound the clinical exam. NSE > 33 ng/ml has a high false-positive rate in patients treated with hypothermia and should be interpreted with caution.

show abstract

Section: Discussionmentioning

confidence: 90%

Predictors of neurologic outcome in hypothermia after cardiac arrest

Fugate

Wijdicks²,

Mandrekar³

et al. 2010

Annals of Neurology

306

206

View full text Add to dashboard Cite

show abstract

“…Prognostication is frequently the responsibility of a neurologist or neurointensivist who is designated with the task of helping the family and other healthcare providers make sensible decisions regarding the intensity of care and end-of-life decisions. In addition to serial examinations, ancillary tests (EEG, serum neuron-specific enolase, and somatosensory evoked potentials [8,9]) play an increasing role in the decision-making process and help clinicians and families make these decisions (5,7). Since the study in question focused on EEG, let's also focus on this single aspect of care in patients who have suffered from cardiac arrest.…”

Section: Cold But Not Dead: the Role Of Eeg In Predicting The Outcomementioning

confidence: 99%

Cold but Not Dead: The Role of EEG in Predicting the Outcome after Cardiac Arrest

Szaflarski¹

2016

Epilepsy Curr

View full text Add to dashboard Cite

In Clinical Science CommentaryWith publication of the 2005 American Heart Association guidelines for cardiopulmonary resuscitation, "induced hypothermia protocol" after cardiac arrest has gradually become a standard of care. Specific local guidelines and care pathways were developed to help with implementation and to streamline such protocols. These protocols, based on the results of two randomized clinical trials (1, 2), can be paraphrased as induction of mild hypothermia (32-34 o C) for a period of up to 24 hours in any patient who has suffered an out-of-hospital cardiac arrest; slow rewarming to normal temperature occurs thereafter. Continuous EEG monitoring in patients undergoing induced therapeutic hypothermia is also standard, typically initiated at the time of hypothermia induction and continued for up to 24 hours after the completion of rewarming (3,4). However, the role of EEG in prognostication after a hypoxicischemic event is not entirely clear, documented in the 2006 guideline published by the American Academy of Neurology as "generalized suppression to ≤20µV, burst suppression

show abstract

“…A complete record of the EEGs from the initial stage of the disease to the terminal stage of the disease have been successively recorded with regard to 12 patients (Cases 1-5, 7, 9, 11, 13, 15, 18 and 22). Consequently out of 12 cases, 6 patients (Cases 1-3, 5, 7,9) had marked EEG abnormalities from the initial stage that persisted through their terminal stage. All 6 patients were of the PME group and had had a juvenile onset (mean age of onset: 10.7 years).…”

Section: Clinical Course and Eeg Changesmentioning

confidence: 99%

Electroencephalographic Findings of Hereditary Dentatorubral‐Pallidoluysian Atrophy (DRPLA)

Inazuki

Baba

Naito

1989

Psychiatry Clin Neurosci

View full text Add to dashboard Cite

The electroencephalograms (EEGs) of 22 patients suffering from hereditary DRPLA were studied. Epileptoform patterns were observed in 14 patients (63.6%) with epileptic seizures. The epileptoform patterns most frequently observed were those atypical spike‐wave complexes. Slow wave bursts were seen in 18 patients (81.8%). Photosensitivity was revealed in six (27.3%) patients, all of whom presented progressive myoclonus epilepsy (PME) syndrome. Abnormal background activity was evident in 17 (77.3%) patients. These abnormalities in EEG were more severe in patients in the PME type than those of the A (ataxia) and AE (ataxia and epilepsy) types.

show abstract

Electroencephalographic Changes in Acute Cerebral Anoxia From Cardiac or Respiratory Arrest

Cited by 20 publications

References 0 publications

Predictors of neurologic outcome in hypothermia after cardiac arrest

Predictors of neurologic outcome in hypothermia after cardiac arrest

Cold but Not Dead: The Role of EEG in Predicting the Outcome after Cardiac Arrest

Electroencephalographic Findings of Hereditary Dentatorubral‐Pallidoluysian Atrophy (DRPLA)

Contact Info

Product

Resources

About