“…Components of the damaged PMN may, in addition, generate an immune response, and this response could contribute to the vascular pathology of this condition [28]. Changes in lymphocyte populations during the acute phase of invasive amebiasis such as a decreased helper T (CD4) lymphocyte count and increased cytotoxic T (CD8) lymphocytes, as well as a decrease in the proliferative response of T lymphocytes to amebic antigens, also are found [30,31].…”
Neutrophils and lymphocytes, particularly the former, are associated significantly with the density of parasites. Our findings support the theory that PMN interaction with E. histolytica contributes to the pathogenesis of amebic intestinal lesions.
“…Components of the damaged PMN may, in addition, generate an immune response, and this response could contribute to the vascular pathology of this condition [28]. Changes in lymphocyte populations during the acute phase of invasive amebiasis such as a decreased helper T (CD4) lymphocyte count and increased cytotoxic T (CD8) lymphocytes, as well as a decrease in the proliferative response of T lymphocytes to amebic antigens, also are found [30,31].…”
Neutrophils and lymphocytes, particularly the former, are associated significantly with the density of parasites. Our findings support the theory that PMN interaction with E. histolytica contributes to the pathogenesis of amebic intestinal lesions.
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