1996
DOI: 10.1016/s0026-0495(96)90039-6
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Effect of endothelin-1 in man—impact on basal and stimulated concentrations of luteinizing hormone, follicle-stimulating hormone, thyrotropin, growth hormone, corticotropin, and prolactin with and without pretreatment with nifedipine

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Cited by 12 publications
(8 citation statements)
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“…This is further supported by the tendency found in our study for higher values of ET in patients with hypogonadotrophic hypogonadism, in whom gonadotrophin concentrations are low. There are several lines of evidence that point to endothelins participating in the regulation of pituitary hormone secretion (Vierhapper et al ., 1993; Vierhapper, 1996; Kiefer et al ., 2000). Release of LH and FSH is stimulated by ET‐3 (Kanyicska et al ., 1991; Furudate et al ., 1994; Naruse et al ., 1994).…”
Section: Discussionmentioning
confidence: 99%
See 1 more Smart Citation
“…This is further supported by the tendency found in our study for higher values of ET in patients with hypogonadotrophic hypogonadism, in whom gonadotrophin concentrations are low. There are several lines of evidence that point to endothelins participating in the regulation of pituitary hormone secretion (Vierhapper et al ., 1993; Vierhapper, 1996; Kiefer et al ., 2000). Release of LH and FSH is stimulated by ET‐3 (Kanyicska et al ., 1991; Furudate et al ., 1994; Naruse et al ., 1994).…”
Section: Discussionmentioning
confidence: 99%
“…Like gonadotrophin releasing hormone (GnRH), ET‐1 stimulates, in vitro , luteinizing hormone (LH) and follicle‐stimulating hormone (FSH) production from the pituitary (Stojilkovic et al . 1992; Vierhapper, 1996) and has a modulatory effect on prolactin release (Samson et al ., 1991; Vierhapper, 1996). Hammami et al .…”
Section: Introductionmentioning
confidence: 99%
“…It has been reported that ET-1 given i.v. augmented basal and CRH-stimulated ACTH release in normal human subjects (Vierhapper et al 1993), possibly because of increased blood pressure associated with ET-1 infusion, since both the increased ACTH and blood pressure were reversed by the calcium channel antagonist nifedipine (Vierhapper 1996). These data are difficult to interpret, since a cocktail dose of all synthetic hypothalamic releasing hormones was given to each subject, and multiple confounding interactions at the anterior pituitary cannot be discounted.…”
Section: Endothelins (Ets)mentioning
confidence: 99%
“…Another possibility is the involvement of growth hormone (GH), which inhibits ET-1 action in human endothelial cells [44]. However, intravenous ET-1 injection suppresses the growth hormone-releasing hormone (GH-RH)-stimulated increase in GH secretion in healthy man [45]. This suggests the feedback system between the ET-1 system and the GH system.…”
Section: Discussionmentioning
confidence: 99%