Effect of Caffeine on LT, VT and HRVT

Karapetian, Gregory K.; Engels, Hermann J.; Gretebeck, Kimberlee A.; Gretebeck, Randall J.

doi:10.1055/s-0032-1301904

Cited by 49 publications

(42 citation statements)

References 29 publications

(57 reference statements)

Supporting

Mentioning

Contrasting

Unclassified

Order By: Relevance

“…Multiple potentially confounding factors can affect HRV, including age, cardiovascular disease, diabetes, renal failure, obstructive sleep apnea, tobacco, caffeine, and drugs [34–36]. Although few patients in this study had such obvious features, cardiovascular disease and prediabetes are often subclinical in SLE [37, 38], so further evaluation of these potential confounders is in order.…”

Section: Discussionmentioning

confidence: 99%

Impact of heart rate variability, a marker for cardiac health, on lupus disease activity

et al. 2016

View full text Add to dashboard Cite

show abstract

Section: Discussionmentioning

confidence: 99%

Impact of heart rate variability, a marker for cardiac health, on lupus disease activity

et al. 2016

View full text Add to dashboard Cite

show abstract

“…Furthermore, during exercise the response of these measures is generally consistent with current understandings of cardiac autonomic regulation, i.e., an increase in cSNA and a decrease in cPNA (Robinson et al, 1966; Rowell and Oleary, 1990; White and Raven, 2014). Under normal conditions these measures are gradually reduced during progressive exercise (Yamamoto et al, 1991; Tulppo et al, 1996, 1998; Hautala et al, 2003; Casties et al, 2006; Martinmaki et al, 2008; Karapetian et al, 2012), whereas this response is greatly reduced or abolished under parasympathetic blockade (Tulppo et al, 1996; Warren et al, 1997; Hautala et al, 2003). …”

Section: What Is Hrv and How Is It Quantified?mentioning

confidence: 99%

“…In addition to different modalities being utilized between these studies, the duration for which participants exercised at each intensity varies greatly, such as 2 min (Tulppo et al, 1996; Martinmaki et al, 2008), 3 min (Tulppo et al, 1998, 1999; Hautala et al, 2003; Karapetian et al, 2012), 5 min (Radaelli et al, 1996), 8 min (Casties et al, 2006; Michael et al, 2016), 10 min (Martinmaki and Rusko, 2008) and 15 or more min (Yamamoto et al, 1991; Saito and Nakamura, 1995; Leicht et al, 2008; Boettger et al, 2010). Nevertheless, an analysis of the literature allows us to identify some general responses for some HRV measures as a function of exercise intensity.…”

Section: Hrv During Exercisementioning

confidence: 99%

“…Several studies have reported that higher exercise intensity is associated with a lower SDRR (Yamamoto et al, 1991; Saito and Nakamura, 1995; Radaelli et al, 1996; Tulppo et al, 1996; Hautala et al, 2003; Casties et al, 2006; Spadacini et al, 2006; Leicht et al, 2008; Fisher et al, 2009; Karapetian et al, 2012). Compared to resting values which vary greatly (typically 40–100 ms), there is a somewhat consistent intensity dose-response, namely a curvilinear decay to 3–10 ms for exercise >160 b.min −1 .…”

Section: Hrv During Exercisementioning

confidence: 99%

See 1 more Smart Citation

Cardiac Autonomic Responses during Exercise and Post-exercise Recovery Using Heart Rate Variability and Systolic Time Intervals—A Review

2017

View full text Add to dashboard Cite

Cardiac parasympathetic activity may be non-invasively investigated using heart rate variability (HRV), although HRV is not widely accepted to reflect sympathetic activity. Instead, cardiac sympathetic activity may be investigated using systolic time intervals (STI), such as the pre-ejection period. Although these autonomic indices are typically measured during rest, the “reactivity hypothesis” suggests that investigating responses to a stressor (e.g., exercise) may be a valuable monitoring approach in clinical and high-performance settings. However, when interpreting these indices it is important to consider how the exercise dose itself (i.e., intensity, duration, and modality) may influence the response. Therefore, the purpose of this investigation was to review the literature regarding how the exercise dosage influences these autonomic indices during exercise and acute post-exercise recovery. There are substantial methodological variations throughout the literature regarding HRV responses to exercise, in terms of exercise protocols and HRV analysis techniques. Exercise intensity is the primary factor influencing HRV, with a greater intensity eliciting a lower HRV during exercise up to moderate-high intensity, with minimal change observed as intensity is increased further. Post-exercise, a greater preceding intensity is associated with a slower HRV recovery, although the dose-response remains unclear. A longer exercise duration has been reported to elicit a lower HRV only during low-moderate intensity and when accompanied by cardiovascular drift, while a small number of studies have reported conflicting results regarding whether a longer duration delays HRV recovery. “Modality” has been defined multiple ways, with limited evidence suggesting exercise of a greater muscle mass and/or energy expenditure may delay HRV recovery. STI responses during exercise and recovery have seldom been reported, although limited data suggests that intensity is a key determining factor. Concurrent monitoring of HRV and STI may be a valuable non-invasive approach to investigate autonomic stress reactivity; however, this integrative approach has not yet been applied with regards to exercise stressors.

show abstract

“…Assessment of cardiac autonomic nervous system modulation during an incremental stress test or during the recovery phase has been considered a promising strategy for risk stratification and exercise prescription for different populations 1,2,3,4,5 . As established, during exercise there is reciprocity in heart rate modulation between sympathetic hyperactivity and parasympathetic inhibition 6,7,8 ; short-term, post-exercise cardiovascular adaptation is associated with a simultaneous rapid increase in parasympathetic and decrease in sympathetic activity 9,10 .…”

Section: Introductionmentioning

confidence: 99%

Reliability of heart rate variability threshold and parasympathetic reactivation after a submaximal exercise test

Cruz

Rolim

Pires

et al. 2017

Motriz: rev. educ. fis.

View full text Add to dashboard Cite

-The objective of this study was to evaluate reproducibility of heart rate variability threshold (HRVT) and parasympathetic reactivation in physically active men (n = 16, 24.3 ± 5.1 years). During the test, HRVT was assessed by SD1 and r-MSSD dynamics. Immediately after exercise, r-MSSD was analyzed in segments of 60 seconds for a period of five minutes. High absolute and relatively reproducible analysis of HRVT were observed, as assessed by SD1 and r-MSSD dynamics (ICC = 0.92, CV = 10.8, SEM = 5.8). During the recovery phase, a moderate to high reproducibility was observed for r-MSSD from the first to the fifth minute (ICC = 0.69-0.95, CV = 7.5-14.2, SEM = 0.07-1.35). We conclude that HRVT and r-MSSD analysis after a submaximal stress test are highly reproducible measures that might be used to assess the acute and chronic effects of exercise training on cardiac autonomic modulation during and/or after a submaximal stress test.

show abstract

Effect of Caffeine on LT, VT and HRVT

Abstract: These data demonstrate the robustness of the lactate, ventilatory and heart rate variability thresholds when challenged by a physiological dose of caffeine.

Cited by 49 publications

References 29 publications

Impact of heart rate variability, a marker for cardiac health, on lupus disease activity

Impact of heart rate variability, a marker for cardiac health, on lupus disease activity

Cardiac Autonomic Responses during Exercise and Post-exercise Recovery Using Heart Rate Variability and Systolic Time Intervals—A Review

Reliability of heart rate variability threshold and parasympathetic reactivation after a submaximal exercise test

Contact Info

Product

Resources

About