Ectopic Gastric Mucosa in Proximal Esophagus

DeRidder, Peter H.; Shah, Kuldeep

doi:10.1097/00004836-198610000-00003

Cited by 32 publications

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“…Heterotopic gastric mucosa in the upper oesophagus is also known as an inlet patch and has a prevalence at endoscopy estimated at 2 -4 % [1]. These lesions are usually asymptomatic and clinically unimportant [2,3], but are occasionally symptomatic with dysphagia or heartburn or they may rarely ulcerate, perforate or bleed [4,5].…”

mentioning

confidence: 99%

Symptomatic Ulceration of an Acid-Producing Oesophageal Inlet Patch Colonized by Helicobacter Pylori

et al. 2002

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mentioning

confidence: 99%

Symptomatic Ulceration of an Acid-Producing Oesophageal Inlet Patch Colonized by Helicobacter Pylori

et al. 2002

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“…Heterotopic gastric mucosa in the upper oesophagus is considered to be congenital in origin, islands of gastric type mucosa being left in the proximal oesophagus during embryogenesis (Gray and Skundalakis, 1972;Jabbari et al, 1985;Shah et al, 1986). However, it has been suggested that in some cases it represents a reparative error in the presence of chronic inflammation as occurs in Barrett's oesophagus (Rattner and McKinley, 1986).…”

Section: Discussionmentioning

confidence: 99%

Symptomatic heterotopic gastric mucosa in the upper oesophagus

1998

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“…As per the literature, IPs are generally found between the first 3 cm and the upper esophageal sphincter, on the lateral or posterior wall and sometimes encroaching on the upper sphincter. 10…”

Section: Methodsmentioning

confidence: 99%

Heterotopic Gastric Mucosa in the Proximal Esophagus (Inlet Patch): Endoscopic Prevalence, Clinico-pathological Characteristics and Its Association with Helicobacter pylori

Rathod

Anand

Ramesh

et al. 2022

Journal of Digestive Endoscopy

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Objective To determine the prevalence of the inlet patch (IP), its clinico-pathological features, and its association with Helicobacter pylori. Materials and Methods A prospective observational study was performed on 1,889 patients referred for esophagogastroduodenoscopy for various reasons, primarily for the evaluation of dyspepsia. All patients were enquired about the presence of symptoms and carefully examined for the presence of IP during upper gastrointestinal (GI) endoscopy. Biopsies were taken from the patients who had IP. Statistical Analysis All statistical analyses were performed using the Statistical Package for Social Sciences (SPSS) 13.0 software for Windows XP. Categorical variables were compared using the chi-squared test or Fisher's exact test and continuous variables were compared using Student's t-test and univariate analysis. A P-value of less than 0.05 was considered to be statistically significant. Results Inlet patches were found in 34 of 1,889 patients (1.8%). H. pylori was identified in 23.52% of patients (8/34) with IP. Gastric H. pylori infection was positive in all (08/08) patients who had IP. Colonization of H. pylori was more common in antral type mucosa (6/8). H. pylori positivity in the IP correlated with globus sensation symptom in our study, 87.5% of patients with IP and H. pylori positive had globus sensation. Conclusion The prevalence of IP seems to be underestimated. H. pylori colonization of the IP is common and it positively correlates with globus sensation and is closely related to the H. pylori density in the stomach. Though preneoplasia within IP is rare, which does not support the recommendation to regularly obtain biopsies for histopathology, it might be beneficial in a subset of patients with persistent globus sensation.

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Ectopic Gastric Mucosa in Proximal Esophagus

Cited by 32 publications

References 0 publications

Symptomatic Ulceration of an Acid-Producing Oesophageal Inlet Patch Colonized by Helicobacter Pylori

Symptomatic Ulceration of an Acid-Producing Oesophageal Inlet Patch Colonized by Helicobacter Pylori

Symptomatic heterotopic gastric mucosa in the upper oesophagus

Heterotopic Gastric Mucosa in the Proximal Esophagus (Inlet Patch): Endoscopic Prevalence, Clinico-pathological Characteristics and Its Association with Helicobacter pylori

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