Duplication of the Ileum with Heterotopic Gastric Mucosa Pseudomyxoma Peritonei and Nonrotation of the Midgut

Duffy, G.; Enríquez, A. Almazan; Watson, W.C.

doi:10.1016/s0016-5085(19)32901-4

Cited by 14 publications

(3 citation statements)

References 12 publications

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“…A congenital type of aberrant gastric gland-that is, gastric heterotopia in the intestine-has been frequently encountered in patients with Meckel's diverticulum, less frequently with intestinal duplication (Ripstein, 1949;Stockman et al, 1960;Duffy et al, 1974) and other miscellaneous conditions (Lee et al, 1970), while the acquired type of gastric gland-that is, gastric metaplasia-has rarely been found. Several authors have reported on pyloric gland metaplasia in various intestinal disorders (Liber, 1951;Antonius et al, 1960) and a few authors have reported on gastric metaplasia in the duodenum (James, 1964;Hoedemaeker, 1970).…”

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Gastric gland metaplasia in the small and large intestine.

Yokoyama¹,

Kozuka²,

Ito³

et al. 1977

Gut

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SUMMARY Fifty-six surgical specimens with various ulcerative intestinal disorders were microscopically investigated for evidence of gastric gland metaplasia. Thirty-one specimens (55.4%) showed pyloric gland metaplasia. Among the 31 patients with pyloric gland metaplasia, five showed true gastric metaplasia, consisting of parietal cells, chief cells, and mucous neck cells. The percentage of true gastric metaplasia among pyloric gland metaplasia was as high as 16 %, an overall frequency of 9% among various ulcerative intestinal disorders. The mechanism of pyloric gland metaplasia and true gastric metaplasia is not understood, but may occur secondary to submucosal response to ulcer healing and subsequent alteration of the intraluminal condition in the intestine.A congenital type of aberrant gastric gland-that is, gastric heterotopia in the intestine-has been frequently encountered in patients with Meckel's diverticulum, less frequently with intestinal duplication (Ripstein, 1949;Stockman et al., 1960;Duffy et al., 1974) and other miscellaneous conditions (Lee et al., 1970), while the acquired type of gastric gland-that is, gastric metaplasia-has rarely been found. Several authors have reported on pyloric gland metaplasia in various intestinal disorders (Liber, 1951;Antonius et al., 1960) and a few authors have reported on gastric metaplasia in the duodenum (James, 1964;Hoedemaeker, 1970 mucous neck cells among 31 cases which exhibited pyloric gland metaplasia in the small and large intestine. MethodsA series of 56 specimens of intestinal disorders showing benign ulcerative lesions was investigated for evidence of gastric metaplasia. All the surgical specimens were selected from patients who were admitted from 1965 to 1975 to the Second Department of Surgery at Nagoya University School of Medicine and the Yokoyama Gastrointestinal Hospital, Nagoya, Japan. Neither biopsy specimen nor post mortem materials was included in this study.In the interpretation of histological lesions, the term 'pyloric gland metaplasia' was used for glands resembling those of the pyloric region, and the term 'true gastric metaplasia' for glands consisting of parietal cells, chief cells, and mucous neck cells. ResultsOf the total 56 specimens, pyloric gland metaplasia of varying degrees was demonstrated in 31 (54 4 %) as shown in the

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confidence: 99%

Gastric gland metaplasia in the small and large intestine.

Yokoyama¹,

Kozuka²,

Ito³

et al. 1977

Gut

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“…Various theories proposed regarding the development of DC include (a) recanalization after the solid epithelial stage of embryonic bowel development; (b) intrauterine vascular accident theory; (c) persistent embryologic diverticula, but no single theory can explain all the known duplications [ 2 – 4 ]. The accepted diagnostic criteria for an enteric DC include three characteristics—a well-developed smooth muscle coat, epithelial lining representing some portion of the AT mucosa, and intimate anatomic association with some portion of the AT [ 5 ]. However, cysts with no discernible communication or connection to the adjacent AT but with presence of typical histopathological features of DC would qualify for a diagnosis of an isolated DC [ 6 ].…”

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confidence: 99%

“…Regional and mesenteric LN metastasis. Developed carcinomatosis after 8 months following surgery and chemotherapy Duffy G et al [ 5 ] 1974 19 M Recurrent acute AP Ileum PMP. Ileal tubular DC.…”

Section: Discussionmentioning

confidence: 99%

Adenocarcinoma of Jejunal Duplication Cyst—Case Report and Review of Literature

Viswanath

Medappil

Rajan

et al. 2021

Indian J Surg Oncol

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Alimentary tract duplication cyst refers to congenital malformations that involve the mesenteric side of bowel and share a common wall or blood supply with the bowel. Duplication cyst occurs in any segment of the gastrointestinal tract from mouth to anus, but is more commonly seen in the mesentery of the ileo-colic region. Duplication cyst presents more frequently in children with abdominal pain or swelling. In adults, the diagnosis of duplication cyst is incidental and can present with complications like bleed, fistula formation, inflammatory mass, and rarely malignant transformation. English-language literature review has shown only three cases of malignancies reported so far associated with jejunal duplication cyst. A case of incidentally detected metastatic adenocarcinoma in a jejunal duplication cyst is being reported here, along with literature review of malignancies associated with small bowel duplication cyst.

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