Duodenal Obstruction

Sweed, Yechiel

doi:10.1007/978-3-540-69560-8_39

Cited by 6 publications

(21 citation statements)

References 3 publications

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“…An early diagnosis is important because of the high risk of a life-threatening midgut volvulus [4]. The most common presentation of malrotation is bilious vomiting due to duodenal obstruction from intermittent midgut volvulus rather than compression by Ladd’s bands or kinking of the duodenum [4].…”

Section: Discussionmentioning

confidence: 99%

Persistent Nonbilious Vomiting in a Child: Possible Duodenal Webbing

et al. 2017

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An association between malrotation and congenital duodenal webbing is rare. We present our experience with four patients at two centers, and a review of published reports. There are currently 94 reported cases of duodenal pathology associated with malrotation. However, only 15 of the 94 cases (15.9%) include patients with malrotation and a duodenal web. We suggest that nonbilious vomiting in a child must prompt the surgeon to consider duodenal pathology even in the presence of malrotation.

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Section: Discussionmentioning

confidence: 99%

Persistent Nonbilious Vomiting in a Child: Possible Duodenal Webbing

et al. 2017

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“…While there are several researchers that recommend proving intestinal continuity during the repair of duodenal atresia, 1–5 there are several additional researchers who make no such suggestion. 9,12–16 This suggestion may be borrowed from experience with exploration for a known jejunoileal atresia where the possible presence of multiple atresias is well documented.…”

Section: Discussionmentioning

confidence: 99%

“…1–5 Therefore, the laparoscopic approach has been criticized for its limited ability to prove complete intestinal continuity. However, duodenal atresia and jejunoileal atresias do not share common embryologic origins.…”

Section: Introductionmentioning

confidence: 99%

Should We Be Concerned About Jejunoileal Atresia During Repair of Duodenal Atresia?

Peter

Little

Barsness

et al. 2010

Journal of Laparoendoscopic & Advanced Surgical Techniques

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Introduction During repair for duodenal atresia, it has been emphasized that inspection of the small bowel to identify a second atresia is required. The laparoscopic approach for repair of duodenal atresia has been criticized for its limitation to perform this step. Given that duodenal atresia and jejunoileal atresias do not share common embryologic origins, we question the validity of this concern. Therefore, we conducted a multicenter retrospective review of duodenal atresia patients to quantify the incidence of jejunoileal atresia in this population. Methods After institutional review board approval (IRB #07-12-187X), a retrospective review was conducted on all patients who have undergone duodenal atresia repair at seven institutions over the past 7–12 years. Demographics and the presence or absence of a jejunoileal atresia were recorded. Results Four hundred eight patients with duodenal atresia were identified. The mean gestaational age was 36.3 ± 2.9 weeks, and the mean weight was 2.5 ± 0.8 kg. Mean age at operation was 19 days (range, 1–1314). There was a 28% incidence of trisomy 21. Two patients (0.5%) were identified as having a second intestinal atresia, and both were type IIIb. One patient was diagnosed at the time of duodenal atresia repair; the other was a delayed diagnosis. Both patients did well after repair. Conclusions In this, the largest series of duodenal atresia patients compiled to date, the rate of a concomitant jejunoileal atresia is less than 1%. This low incidence is not high enough to mandate extensive inspection of the entire bowel in these patients, and a second atresia should not be a concern during laparoscopic repair of duodenal atresia.

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“…There is a high incidence (approximately 50%) of associated anomalies in patients with intrinsic DO, especially Down syndrome which occurs in about 30% of these patients (Sweed 2011;Puri 1981;Young and Wilkinson 1968). Table 1 presents the overall prevalence and distribution of associated anomalies of duodenal atresia.…”

Section: Associated Malformationsmentioning

confidence: 99%

“…Although the annular pancreas forms a constricting ring around the second part of the duodenum (Fig. 1), it is not believed to be the cause of DO (Elliott et al 1968;Escobar et al 2004), and there is usually an associated atresia or stenosis in patients with an ST stomach, DU duodenum, AP annular pancreas (Sweed 2009). (The picture added with the courtesy of Prof. Bronshtein Moshe) annular pancreas (Girvan and Stephens 1974;Grosfeld et al 1979;Papandreou et al 2004).…”

Section: Introductionmentioning

confidence: 99%