Dominant Language Functions of the Right Hemisphere?

Ross, Elliott D.; Mesulam, Marek Marsel

doi:10.1001/archneur.1979.00500390062006

Cited by 607 publications

(195 citation statements)

References 14 publications

Supporting

Mentioning

180

Contrasting

Unclassified

Order By: Relevance

“…Author Manuscript facial expressions and subjective experience can act independently (e.g., Ross & Mesulam, 1979). This suggests the need to systematically examine both the subjective experience and the outward expression of emotion.…”

Section: Author Manuscript Author Manuscriptmentioning

confidence: 99%

Emotional experience and expression in schizophrenia and depression.

Berenbaum¹,

Oltmanns²

1992

Journal of Abnormal Psychology

388

250

View full text Add to dashboard Cite

The emotional responses of schizophrenic, depressed, and normal subjects and whether differences in the emotional responding of these groups depended on how emotional responses were elicited or measured were examined. Twenty-three blunted and 20 nonblunted schizophrenics, 17 unipolar depressed subjects, and 20 normal subjects were exposed to a series of affect-eliciting stimuli. The stimuli varied in valence (positive vs. negative) and in level of cognitive demand. Subjects reported their subjective experiences, and their facial expressions were videotaped. Blunted schizophrenics were the least facially expressive, although their reported subjective experiences did not differ from those of the other groups. The nonblunted schizophrenics were more responsive than the depressed subjects to the positive stimuli, although the two groups did not differ in their clinical ratings of affective flatness. Historically, psychopathologists have placed a great deal of emphasis on the role of emotional disturbance in schizophrenia. Whereas both Bleuler (1911/1950) and Kraepelin (1919/1971) considered affective flatness to be a universal symptom of schizophrenia, the International Pilot Study of Schizophrenia (World Health Organization, 1973) revealed that only 66% of the schizophrenic patients studied exhibited flat affect. People with major depression have also been found to have flat affect (e.g., Andreasen, 1979; Pogue-Geile & Harrow, 1984). Although blunted affect may not be a universal symptom of schizophrenia and is not specific to the disorder, it does appear to have prognostic importance (Carpenter,

show abstract

Section: Author Manuscript Author Manuscriptmentioning

confidence: 99%

Emotional experience and expression in schizophrenia and depression.

Berenbaum¹,

Oltmanns²

1992

Journal of Abnormal Psychology

388

250

View full text Add to dashboard Cite

show abstract

“…This does not necessarily correlate with a diminished or inappropriate subjective experience of emotion. Neurological evidence indicated that the systems controlling subjective experience and outward expressions of emotion can operate independently (e.g., Ross and Mesulam 1979). Therefore, overt expressions of mirth (smiles and laughter) were observed in addition to ratings of funniness that the children delivered after watching each film clip.…”

Section: Introductionmentioning

confidence: 99%

From Ice Age to Madagascar: Appreciation of slapstick humor in children with Asperger's syndrome

Weiss

Gschaidbauer²,

Samson

et al. 2013

Humor

View full text Add to dashboard Cite

Abstract:The aim of the present study was to examine whether children with Asperger's syndrome differ from typically developing children in the appreciation of and behavioral responses to simple slapstick type humor, in which cognitive requirements that are commonly impaired in autism spectrum disorders are reduced to a minimum. Short slapstick scenes and matched non-humorous control scenes were extracted from popular movies to produce an appropriate humor assessment material. Twenty-four boys with Asperger's syndrome (5 to 14 years) and 24 age-matched typically developed controls were tested. The results indicated that children with Asperger's syndrome enjoy humorous material as much as healthy children do, if the humor elements are simple and the incongruence can be perceived independently from theory of mind requirements, inferential demands, or language abilities. However, similar funniness ratings and behavioral expressions of mirth to the humorous scenes, but relatively higher values in response to the non-humorous scenes, suggested that the autistic children did not discriminate non-humorous from humorous stimuli as sensitively as the typically developing children did. Moreover, in autistic children, the outwards displays of emotion did not match their reports of subjective amusement. This dissociation may relate to the social interaction and communication difficulties in autism spectrum disorders.

show abstract

“…A correlation has been established among specific cortical areas involved in emotion identification from facial expression (right and left fusiform face area, frontal eye), emotion processing (right entorhinal cortex) and socially relevant information (left temporal pole) [95]. According to a model of Ross and Mesulam, affective prosody comprehension could be related to the right-sided perisylvian region, whose structure is similar to the language areas in the left hemisphere [96]. Later studies have suggested an involvement of the basal ganglia and prefrontal cortex (PFC) [97,98].…”

Section: Social Brainmentioning

confidence: 99%

Social Cognition Impairments in Patients with Multiple Sclerosis and Comparison with Imaging Studies, Disease Duration and Grade of Disability

Ignatova¹,

Todorova²,

Surchev³

2016

Trending Topics in Multiple Sclerosis

View full text Add to dashboard Cite

Cognitive impairments in multiple sclerosis (MS) are heterogeneous and their rate varies between 43% and 70%. A less studied aspect of cognition is social cognition, which is not a uniform theoretical construct. It includes emotion perception, prosody, empathy, theory of mind (ToM) and assessment of mood. In addition to progressive physical disability, social cognitive impairments are a reason for job loss in 24-80% of patients with MS, increased divorce rate, dissolution of partnerships and social communication difficulties.Social cognitive impairments are the result of disruptions in the mentalization network at the neuroanatomical, neurochemical and/or genetic level, which can lead to malfunctions in the dopaminergic-serotoninergic system and to compromising the development of neuroanatomical targets within the network. The wide dissemination of demyelinating lesions and cortical thinning typical of patients with MS often lead to anatomic and functional disorders of the above-mentioned specific brain structures. A correlation has been established among specific cortical areas involved in emotion identification from facial expression (right and left fusiform face area, frontal eye), emotion processing (right entorhinal cortex) and socially relevant information (left temporal pole). The most active brain region involved in social cognitive processing is the medal frontal cortex (MFC), which is described as the brain's social cognitive center. During performance of various sociocognitive tasks for mental state attribution, common areas of increased activation in the medial prefrontal gyrus and the temporoparietal junctions are registered bilaterally, while the area of the medial prefrontal cortex (the paracingulate cortex) is the only region uniquely activated by the performance of ToM.Most MRI studies of patients with MS found a correlation between the cognitive and/or affective disorders, on the one hand, and lesion localization, total lesion load, or cerebral atrophy, on the other. A significant correlation was also reported between © 2016 The Author(s). Licensee InTech. This chapter is distributed under the terms of the Creative Commons Attribution License (http://creativecommons.org/licenses/by/3.0), which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited.abnormal activities in specific cortical zones on fMRI, on the one hand, and clinical manifestation, conventional MRI findings and behavioral changes, on the other. Altered patterns of brain activity were found in all clinical phenotypes of MS, including when cognitive abilities were intact and/or restored, social cognitive dysfunction in MS actually affects all stages of the disease and all types of clinical course. The cortical plastic changes are a dynamic phenomenon that can be modulated by external factors. This phenomenon would facilitate the mapping of individual strategies for adequate treatment and rehabilitation of each patient.

show abstract

Dominant Language Functions of the Right Hemisphere?

Cited by 607 publications

References 14 publications

Emotional experience and expression in schizophrenia and depression.

Emotional experience and expression in schizophrenia and depression.

From Ice Age to Madagascar: Appreciation of slapstick humor in children with Asperger's syndrome

Social Cognition Impairments in Patients with Multiple Sclerosis and Comparison with Imaging Studies, Disease Duration and Grade of Disability

Contact Info

Product

Resources

About