2003
DOI: 10.1046/j.1526-4610.2003.03041.x
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Does Chronic Daily Headache Arise De Novo in Association With Regular Use of Analgesics?

Abstract: These findings suggest that individuals with primary headache, specifically migraine, are predisposed to developing chronic daily headache in association with regular use of analgesics.

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Cited by 259 publications
(193 citation statements)
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“…Migraine patients are particularly predisposed to develop chronic headache in association with medication overuse [8]. The regular use of symptomatic medications is associated with the development of chronic headache in individuals who have an existing history of migraine; medication overuse per se did not cause the development of headache de novo in patients without previous headache history.…”
Section: Medication Overusementioning
confidence: 99%
“…Migraine patients are particularly predisposed to develop chronic headache in association with medication overuse [8]. The regular use of symptomatic medications is associated with the development of chronic headache in individuals who have an existing history of migraine; medication overuse per se did not cause the development of headache de novo in patients without previous headache history.…”
Section: Medication Overusementioning
confidence: 99%
“…Medication-overuse headache is a major health problem [10]. Patients with migraine and tension-type headache have a higher potential for medicationoveruse headache [11], and relapses of migraine occur in migraineurs who have been placed on analgesics for other ailments. On the contrary, chronic overuse of analgesics does not cause increased headache in non-migraineurs [12,13].…”
Section: Discussionmentioning
confidence: 99%
“…14,120 Within this group of MOH patients, the vast majority first present with episodic migraine or tension-type headache 121 as opposed to other forms of primary headache such as cluster headache. 122 We hypothesise this selective propensity to develop MOH stems from altered pro-inflammatory central immune signalling in patients with migraine and tension-type headache, or the presence of underlying central sensitisation, which renders them particularly susceptible to the effects of opioid-induced glial activation.…”
Section: Pro-inflammatory Central Immune Signalling Hypothesis Of Mohmentioning
confidence: 99%