Detection of Gallstones in Acute Pancreatitis: When and How?

Johnson, C. D.; Lévy, Philippe

doi:10.1159/000224147

Cited by 29 publications

(15 citation statements)

References 29 publications

(36 reference statements)

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“…However, by excluding those with a previous history of exocrine pancreatic disease this possibility was reduced. Also, the definition of gallstone-related—and non-gallstone-related—acute pancreatitis is in the focus of an ongoing discussion 26. Finally, we could not adjust our analyses for the occurrence of other known risk factors of non-gallstone-related acute pancreatitis: for example, cystic fibrosis, hypercalcaemia, hypertriglycaemia and drug-induced pancreatitis 1.…”

Section: Discussionmentioning

confidence: 99%

Vegetables, fruit and risk of non-gallstone-related acute pancreatitis: a population-based prospective cohort study

Oskarsson

Sadr–Azodi

Orsini

et al. 2012

Gut

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Section: Discussionmentioning

confidence: 99%

Vegetables, fruit and risk of non-gallstone-related acute pancreatitis: a population-based prospective cohort study

Oskarsson

Sadr–Azodi

Orsini

et al. 2012

Gut

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“…The severity of the inflammation, hemorrhage, or necrosis of the pancreas, peripancreatic fluid collection, or abscess are closely related with prognosis [1–3]. Bile reflux into the common bile duct and obstruction of the Ampulla Vater by biliary sludge or stone are supposed to cause biliary pancreatitis [4].…”

Section: Introductionmentioning

confidence: 99%

“…of these ducts are effective on the prognosis and determine the management. If the obstruction persists more than 48 hours, the complications increase [3, 5–7]. The stones in the distal common bile duct and the pancreatic duct could not be detected in all patients with biliary pancreatitis.…”

Section: Introductionmentioning

confidence: 99%

The Predictivity of Serum Biochemical Markers in Acute Biliary Pancreatitis

Güngör

Çağlayan

Polat

et al. 2011

ISRN Gastroenterology

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Background and Aim. There are no accurate methods of differentiating acute biliary pancreatitis. Obstructions of biliary ducts, idiopathic pancreatitis may be related with biliary origin which needs identification for acute treatment. We searched for the predictivity of biochemical markers in early acute biliary pancreatitis. Patients and Methods. Serum levels of AST (Aspartate Transaminase),ALT (Alanine Transaminase), ALP (Alkaline Phosphatase), GGT (Gamma Glutamyl Transferase), total bilirubin, direct bilirubin, LDH (Lactate Dehydrogenase), amylase, lipase, CRP (C-Reactive Protein) and WBC (White Blood Cell) were measured in 157 patients with acute pancreatitis. Biliary and nonbiliary pancreatitis were differentiated by Magnetic Resonance Cholangiopancreatography (MRCP), Endoscopic Retrograde Cholangiopancreatography (ERCP), Intraoperative Cholangiopancreatography (IOC). Cut-off points of admission biochemical markers with sensitivity, specifity, positive predictive value and negative predictive value were determined after identification of significant variables. Receiver Operator Curves were plotted for each biochemical marker. Results. Serum Alkaline Phosphatase, total bilirubin, direct bilirubin, amylase and lipase levels were significantly higher in biliary pancreatitis with a positive predictive value of 80.8%, 83.9%, 81.6%, 78.8%, 79.7%. Conclusion. Increased Alkaline Phosphatase,total bilirubin, direct bilirubin, amylase and lipase levels may be used in prediction of biliary pancreatitis.

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“…Our study population was also administered combination of ACE inhibitor and calcium blocker along with beta-blockers and the mean value for this group was found to be 0.55, thus strongly depicting higher potential of this regimen in reducing higher levels of CRP [16]. Another study reported that beta-blocker ACE inhibitors are highly associated with lowering down CRP level [17].…”

Section: Matrix Science Medica (Msm)mentioning

confidence: 59%

Medicinal Efficacy of Antihypertensive Agents for Acute Inflammation; An Associated Risk Factor of Hypertension

Sarwar¹,

Subhani²,

Bilal³

et al. 2017

Matrix sci. medica

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Hypertension is a lasting therapeutic complaint which can be kept in control by changing one's dietary and lifestyle habits. Present study is based on identification of risk of acute pancreatitis which may occur with different doses of atenolol in Pakistani population. About 50% of our study population was administered with β-blockers. The β-blockers significantly decreased the levels of CRP (C-Reactive Protein) of test population. This observation strongly depicts high potential of beta blockers in stabilization of CRP levels and decreasing inflammatory response. In our study the patients were administered with many combinations with beta-blocker and their results showed that these combinations lower the CRP level in hypertensive patients. We conclude that antihypertensive also minimize the inflammation associated hypertension but not completely, as inflammation was reversed only in 67.5% population who were using beta-blocker. When treating hypertension, anti-inflammatory medicine treatment needs to be endorsed to successfully treat complaints and increase the communal and psychosomatic status of the patient.

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Detection of Gallstones in Acute Pancreatitis: When and How?

Cited by 29 publications

References 29 publications

Vegetables, fruit and risk of non-gallstone-related acute pancreatitis: a population-based prospective cohort study

Vegetables, fruit and risk of non-gallstone-related acute pancreatitis: a population-based prospective cohort study

The Predictivity of Serum Biochemical Markers in Acute Biliary Pancreatitis

Medicinal Efficacy of Antihypertensive Agents for Acute Inflammation; An Associated Risk Factor of Hypertension

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