Der zeitliche Verlauf der Milchsäurebildung bei der Muskelkontraktion.

Embden, Gustav; Lehnartz, Emil; Hentschel, H.

doi:10.1515/bchm2.1927.165.4-6.255

Cited by 17 publications

(2 citation statements)

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“…Both researchers believed that lactic acid was the substance that triggered the contraction. Embden (36)(37)(38), however, deduced that the formation of lactic acid was merely an energy-supplying process, an "endo. thermic recharging of a direct source of energy, the exothermic discharging of which would have resulted in muscle contraction."…”

Section: Anaerobic Work and Lactic Acidmentioning

confidence: 99%

Historical Remarks on the Development of the Aerobic-Anaerobic Threshold up to 1966

Hollmann¹

1985

Int J Sports Med

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During the years 1957 to 1963, we introduced the concept of the onset of anaerobic metabolism to measure cardiopulmonary and peripheral aerobic performance capacity. On the basis of bicycle and crank ergometer work with load increments of 3 min duration, we described a point at which the pulmonary ventilation (VE) increases at a greater rate than O2 uptake (VO2). Because the changes of the arterial blood lactate (Laa) and VE coincide we defined this point as the "point of the optimal ventilatory efficiency," identical with the "O2 endurance performance limit," later called "anaerobic threshold" by Wasserman et al.

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Section: Anaerobic Work and Lactic Acidmentioning

confidence: 99%

Historical Remarks on the Development of the Aerobic-Anaerobic Threshold up to 1966

Hollmann¹

1985

Int J Sports Med

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“…Lactate was identified as an indicator of glycolytic activity early last century (Boycott and Haldane 1908;Embden et al 1927;Hill et al 1924) and soon afterwards blood lactate concentration (BLC) became established as a measure of exercise intensity during incremental and constant-workload exercise (Bang 1936;Hill et al 1924).…”

Section: Introductionmentioning

confidence: 99%

Maximal lactate steady state concentration (MLSS): experimental and modelling approaches

2003

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It is assumed that the maximal lactate steady state (MLSS) can be used to establish the highest workload that can be maintained over time without continual blood lactate accumulation. In untrained subjects, and in both elite and junior athletes, MLSS occurs at different blood lactate concentrations (BLC) for different exercise modes. This suggests that MLSS depends on the motor pattern of exercise and may be a function of the relationship between power output per unit muscle mass and the mass of the muscle primarily engaged in the activity. A computer model has been developed that takes account of current theories relating to the effect of exercise on BLC and to the factors that limit oxygen transport to the muscle cell. Simulations using this model support the suggestion that load per unit of engaged muscle mass accounts for task-specific levels of MLSS. Simulated differences in MLSS appear because the MLSS does not necessarily reflect the real maximal equilibrium between lactate formation and utilization, the LLSS. The higher difference between MLSS and LLSS measured in rowing ergometry compared to cycle ergometry seems to indicate a greater task sensitivity of the BLC response to given changes of exercise intensity during rowing. Whether such a difference may be relevant for a deeper understanding of task-specific training strategies remains a matter for further investigation.

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Anaerobic recovery in man

Cerretelli

Ambrosoli

Fumagalli

1975

Europ. J. Appl. Physiol.

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The lactic acid (L.A.) concentration in blood after a 20 sec supramaximal exercise (2.5 times VO2max) has been measured in 4 subjects in the following experimental conditions: a) during the resting period following the supramaximal exercise (rest recovery) and b) during a 3 min exercise at VO2max immediately following the supramaximal effort (exercise recovery). The L.A. concentration in blood has been found to be consistently higher (on the average by 16.9 mg%) in case (b). Since in such condition it may be reasonably assumed that the oxygen taken up by the subject is completely utilized for the exercise, the increase of blood lactate is considered evidence for the occurrence of anaerobic recovery, i.e. of a partial re-synthesis of the high energy phosphate stores of the muscle (GP = ATP + PC) depleted during the supramaximal effort, at the expense of anaerobic glycolysis. From the increase in blood L.A. concentration during the anaerobic recovery period, the amount of L.A. produced has been estimated together with the amount of GP resynthesized. The latter corresponds to 4 to 7 mMoles/kg of muscle, i.e. to about 25% of the average GP concentration in resting human muscle. The finalistic implication of this mechanism is the prompt restoration of the potential maximal power of the muscle even in the absence of O2.

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Der zeitliche Verlauf der Milchsäurebildung bei der Muskelkontraktion.

Cited by 17 publications

References 0 publications

Historical Remarks on the Development of the Aerobic-Anaerobic Threshold up to 1966

Historical Remarks on the Development of the Aerobic-Anaerobic Threshold up to 1966

Maximal lactate steady state concentration (MLSS): experimental and modelling approaches

Anaerobic recovery in man

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