Depletion of the apical endosome in response to viruses and bacterial toxins provides cell-autonomous host defense at mucosal surfaces

Maeda, Kayaho; Zachos, Nicholas C.; Orzalli, Megan H.; Schmieder, Stefanie S.; Chang, Denis; Gwilt, Katlynn Bugda; Doucet, Michèle; Baetz, Nicholas W.; Lee, Sun; Crawford, Sue E.; Estes, Mary K.; Kagan, Jonathan C.; Turner, Jerrold R.; Lencer, Wayne I.

doi:10.1016/j.chom.2021.12.011

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Cited by 6 publications

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“…The mechanism of reovirus superinfection exclusion is unclear. Known mechanisms of superinfection exclusion include antiviral host responses (32,33), inhibition of viral entry through various mechanisms (17,31,59,60), and competition for host resources (29). Given that type 3 reoviruses are sensitive to type 1 interferon (36), antiviral responses seem a likely mechanism for superinfection exclusion.…”

Section: Discussionmentioning

confidence: 99%