Delayed‐type allergy to systemic corticosteroids

Reinhold, Katharina; Schneider, L.; Hunzelmann, N.; Krieg, Thomas; Scharffetter‐­Kochanek, Karin

doi:10.1034/j.1398-9995.2000.00783.x

Cited by 11 publications

(2 citation statements)

References 6 publications

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“…It is generally accepted, however, that the same cross-reactivity patterns observed for topical preparations occur when they are used systemically. Such patterns should be taken into account for choosing an alternative GC molecule in sensitive patients [89][90][91]. As with their topical counterparts, there will be exceptions to systemic steroid cross-reactivity patterns.…”

Section: Preservatives In Gc Topical Preparations Responsible For Amentioning

confidence: 93%

Cross-Reactivity in Cell-Mediated and IgE-Mediated Hypersensitivity to Glucocorticoids

Ventura

Calogiuri²,

Muratore³

et al. 2006

CPD

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In the last few decades, glucocorticoids have received increasing attention for their capability of provoking systemic hypersensitivity reactions, when administered orally, parenterally, or intralesionally, as well as allergic skin and mucosal symptoms, when applied locally to the skin in patients with contact dermatitis or to the mucosa in patients with asthma and/or rhinitis. However, because of their anti-inflammatory and immunosuppressive properties, glucocorticoids are often not suspected of such hypersensitivity reactions. In addition, because glucocorticoids retain their anti-inflammatory potential, even if they act as sensitizers, the signs and symptoms of allergic reactions are not always obvious, particularly when they overlap with those caused by the very diseases glucocorticoids are used to treat. Moreover, interpretation of diagnostic tests, specifically that of patch-test reactions, can be difficult. In this review, particular attention is addressed to the problem of allergenic cross-reactivity among topical and systemic glucocorticoids. We also look at the clinical and practical aspects of both cell-mediated and IgE-mediated hypersensitivity reactions to glucocorticoids and their consequences on anti-inflammatory therapeutic choices.

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Section: Preservatives In Gc Topical Preparations Responsible For Amentioning

confidence: 93%

Cross-Reactivity in Cell-Mediated and IgE-Mediated Hypersensitivity to Glucocorticoids

Ventura

Calogiuri²,

Muratore³

et al. 2006

CPD

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“…On the other hand, systemically administered corticosteroids appear to be an infrequent cause of immunologically mediated reactions (17–21). Among steroid‐induced allergic reactions, the most frequent is the delayed hypersensitivity because of topical sensitization, with an incidence of 2.9–4.1% (22).…”

mentioning

confidence: 99%

Distinct delayed T‐cell response to β‐methasone and penicillin‐G in the same patient

Scala

Giani

Pastore³

et al. 2003

Allergy

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Current Awareness

2001

Pharmacoepidemiology and Drug

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In order to keep subscribers up‐to‐date with the latest developments in their field, John Wiley & Sons are providing a current awareness service in each issue of the journal. The bibliography contains newly published material in the field of pharmacoepidemiology and drug safety. Each bibliography is divided into 19 sections: 1 Books, Reviews & Symposia; 2 General; 3 Anti‐infective Agents; 4 Cardiovascular System Agents; 5 CNS Depressive Agents; 6 Non‐steroidal Anti‐inflammatory Agents; 7 CNS Agents; 8 Anti‐neoplastic Agents; 9 Haematological Agents; 10 Neuroregulator‐Blocking Agents; 11 Dermatological Agents; 12 Immunosuppressive Agents; 13 Autonomic Agents; 14 Respiratory System Agents; 15 Neuromuscular Agents; 16 Reproductive System Agents; 17 Gastrointestinal System Agents; 18 Anti‐inflammatory Agents ‐ Steroidal; 19 Others. Within each section, articles are listed in alphabetical order with respect to author. If, in the preceding period, no publications are located relevant to any one of these headings, that section will be omitted.

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Delayed‐type allergy to systemic corticosteroids

Cited by 11 publications

References 6 publications

Cross-Reactivity in Cell-Mediated and IgE-Mediated Hypersensitivity to Glucocorticoids

Cross-Reactivity in Cell-Mediated and IgE-Mediated Hypersensitivity to Glucocorticoids

Distinct delayed T‐cell response to β‐methasone and penicillin‐G in the same patient

Current Awareness

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