Craniomandibular osteopathy in Doberman Pinschers

Abstract

Two young Doberman Pinschers with painless, firm enlargement of the mandibles were investigated. Craniomandibular osteopathy was diagnosed in each case on the basis of characteristic radiographic and gross and microscopic lesions.

A review of 14 cases of hypertrophic osteopathy (Marie's disease) in horses in the Republic of Ireland

“…It is of interest to note that, although not currently reported in horses, craniomandibular osteopathy is a disease similar to HO in that it involves proliferation of long bones. This disease occurs in young dogs resulting in bilateral osseous proliferation of the mandible, tympanic bullae and less commonly other bones of the head (Watson et al . 1975, 1995; Taylor et al .…”

Surgical management of chronic otitis secondary to craniomandibular osteopathy in three West Highland white terriers

“…Although the majority of clinical reports identify bilateral involvement of the affected bones in CMO, unilateral mandibular involvement has been previously described (Watson et al 1995). The aetiopathogenesis of CMO remains poorly understood and it is likely to be multifactorial (Riser et al 1967, Watson et al 1975. In a case series of 12 Irish setters with canine leukocyte adhesion deficiency, seven developed radiographic signs consistent with CMO, supporting an underlying infectious cause for these changes (Trowald-Wigh et al 2000).…”

“…Implicated agents include viral (canine distemper virus) and bacterial infection (E. coli) (Watson et al 1995). The occurrence of CMO in a diverse range of breeds other than WHWTs also suggests that either there is an enhanced inherited susceptibility to a causative agent or there are multiple causes of the disease (Watson et al 1975, Thompson 2007.…”

“…), although numerous other breeds have been affected sporadically (Watkins & Bradley , Watson et al . , Burk & Broadhurst , Schulz , Hathcock , Taylor et al . , Franch et al .…”

“…Concurrent involvement of other skull and long bone metaphyses is occasionally reported (Riser et al 1967, Dennis et al 1993, Watson et al 1995, Franch et al 1998, Ratterree et al 2011, Thompson et al 2011, Pettitt et al 2012. West Highland white terriers (WHWT), Scottish terriers and Cairn terriers are at increased risk of developing CMO (Munjar et al 1998, LaFond et al 2002, although numerous other breeds have been affected sporadically (Watkins & Bradley 1966, Watson et al 1975, Burk & Broadhurst 1976, Schulz 1978, Hathcock 1982, Taylor et al 1995, Franch et al 1998, Munjar et al 1998, Huchkowsky 2002, Ratterree et al 2011, Thompson et al 2011, Matiasovic et al 2015. Affected dogs are typically presented at 3 to 9 months of age because of a painful jaw swelling, episodic pyrexia, lethargy and inappetance (Riser et al 1967, Dennis et al 1993.…”