2016
DOI: 10.1016/j.jes.2016.08.001
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Corrigendum to “Development and characterization of monoclonal antibodies against human aryl hydrocarbon receptor” [J. Environ. Sci. 39 (2016) 165–174]

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“…Furthermore, the up-regulation of the pro-adhesive genes could be reversed with the presence of AhR antagonist, CH223191. These data supported that the TCDD treatment could activate AhR signaling pathway in SK-N-SH cells, in which AhR protein has been detected previously (Tian et al 2016). Since the presence of dioxin responsive element (DRE) consensus sequences in the promoter region is one of the features of AhR target genes, we checked the putative promoter of certain genes belonging to PCDHG subfamily, which was the most sensitive subfamily of PCDHs to TCDD treatment in this study.…”
Section: Discussionsupporting
confidence: 86%
“…Furthermore, the up-regulation of the pro-adhesive genes could be reversed with the presence of AhR antagonist, CH223191. These data supported that the TCDD treatment could activate AhR signaling pathway in SK-N-SH cells, in which AhR protein has been detected previously (Tian et al 2016). Since the presence of dioxin responsive element (DRE) consensus sequences in the promoter region is one of the features of AhR target genes, we checked the putative promoter of certain genes belonging to PCDHG subfamily, which was the most sensitive subfamily of PCDHs to TCDD treatment in this study.…”
Section: Discussionsupporting
confidence: 86%