Comparative Biochemistry of Detoxification

Smith, J. N.

doi:10.1016/b978-0-12-395547-0.50015-x

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Formation Of Carbonic Acids1

Discussion1

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1969

1999

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Cited by 17 publications

(2 citation statements)

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“…Analysis of the carbonic acid fraction indicated the formation of cis,cis-muconic acid. Earlier it was found that muconic acid is formed in cells from exogenous benzene or phenol via catechol (Smith, 1964). When catechol is cleaved in microorganisms the product is cis,cis-muconic acid, but in the analogous reaction in mammals, only the trans,trans-isomer was detected.…”

Section: Formation Of Carbonic Acidsmentioning

confidence: 99%

Detoxication of Phenol in Annual Plant Seedlings

Ugrekhelidze¹,

Kvesitadze²,

Arziani

et al. 1999

Ecotoxicology and Environmental Safety

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Section: Formation Of Carbonic Acidsmentioning

confidence: 99%

Detoxication of Phenol in Annual Plant Seedlings

Ugrekhelidze¹,

Kvesitadze²,

Arziani

et al. 1999

Ecotoxicology and Environmental Safety

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“…Cassava contains cyanogenic glycosides, and in the preparation of its flour (gari) a substantial amount of the cyanide is removed. Detoxification of the residual cyanide from cassava is known to take place in the kidney and the liver [11]. The rhodanase-mediated incorporation of cyanide with thiosulphate to form thiocyanate is the main detoxification process.…”

Section: Discussionmentioning

confidence: 99%

Effect of gari diet on the pathological changes of the aorta, liver, and kidneys of rats.

Onyeneke

Ononogbu

1989

J. Clin. Biochem. Nutr.

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The effects of feeding of an experimental diet consisting of 16 and 32% of gari by weight were compared with those of the gari-free, control diet in rats. The experiment was carried out for a period of six months during which two rats from each group (A, control; B, 16% gari incorporation; and C, 32% gari incorporation) were killed for histological study. Histological examination of the aorta, liver, and kidneys revealed degenerative changes in group B and C rats, as evidenced by vacuolation and accumulation of periportal fat. These manifestations started at the third month of the experiment. At the fourth month there was massive degeneration of the arterial wall linings followed by development of fatty streaks in the lumen of the arteries.

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