Cognitive-behavioral issues in the treatment and management of chronic daily headache

Lipchik, Gay L.; Nash, Justin M.

doi:10.1007/s11916-002-0066-x

Cited by 22 publications

(9 citation statements)

References 49 publications

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“…Headaches or affective distress may not improve until the comorbid psychopathology improves. 23 Our study supports the view that combined psychologic and pharmacologic treatment need to be considered. 24,25 Relaxation or biofeedback training and short-term cognitive-behavioral therapy are particularly useful.…”

Section: Resultssupporting

confidence: 86%

Psychiatric Comorbidity in Transformed Migraine: Presentation, Treatment, Impact and Outcome

Abbas¹,

Ismaiel²,

Young³

et al. 2010

JJP

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Section: Resultssupporting

confidence: 86%

Psychiatric Comorbidity in Transformed Migraine: Presentation, Treatment, Impact and Outcome

Abbas¹,

Ismaiel²,

Young³

et al. 2010

JJP

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“…Combined first-line drug and behavioral treatments, chiefly relaxation, biofeedback and cognitive-behavioral therapy, are more beneficial in chronic tension-type headache than if administered alone [76]. It is likely that the efficacy of amitriptyline, used as first-line therapy in chronic tension-type headache, could be only partly explained by the blockade of 5-HT reuptake.…”

Section: Discussionmentioning

confidence: 99%

Pain and tension-type headache: a review of the possible pathophysiological mechanisms

Milanov

Богданова

2004

J Headache Pain

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Tension-type headache represents a considerable health problem and is one of the most costly diseases in the modern society. However, the knowledge about the pathophysiological mechanisms leading to this disorder is limited. The review presents a summary of available research and experimental data on the pathophysiological mechanisms and the recent pathophysiological models. Although the pain in tension-type headache clinically resembles pain from myofascial tissues, both peripheral and central mechanisms are believed to be involved. It is likely that myofascial nociception is important in episodic tension-type headache and that central mechanisms are involved in the pathophysiology of the chronic form. Neurophysiological investigations are introduced to be the most suitable to confirm an involvement of trigeminal pathways, to substantiate recent theories on sensitization phenomena, and to disclose the exact pain-control mechanisms. More studies are needed to elucidate the mechanisms leading to central sensitization and to develop new therapeutic strategies.

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“…*Discussion of individual drugs used for the management of chronic orofacial pain is beyond the scope of this review, and we refer the reader to the relevant textbook by Sharav and Benoliel, 2015 effective in the treatment of insomnia secondary to chronically painful medical conditions such as fibromyalgia, mixed chronic pain conditions, back pain, and arthritis and osteoarthritis (Roehrs, 2009;Sharav and Benoliel, 2015).CBT-I strategies not only change sleep patterns, but may also improve pain in adults (Vitiello et al, 2009). CBT-I approaches used for pain management in headaches (Lipchik and Nash, 2002) and TMD (Ferrando et al, 2012) were beneficial. Indeed, poor sleep hygiene contributes to sleep problems among chronic migraine patients (Calhoun et al, 2006), and this was modified by behavioral sleep interventions, which reduced headache frequency and the headache index, and chronic migraine reverted to episodic migraine (Calhoun and Ford, 2007).…”

Section: Behavioral Approachmentioning

confidence: 99%

An update of management of insomnia in patients with chronic orofacial pain

et al. 2017

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In this review, we discuss the management of chronic orofacial pain (COFP) patients with insomnia. Diagnostic work-up and follow-up routines of COFP patients should include assessment of sleep problems. Management is based on a multidisciplinary approach, addressing the factors that modulate the pain experience as well as insomnia and including both non-pharmacological and pharmacological modalities. Parallel to treatment, patients should receive therapy for comorbid medical and psychiatric disorders, and possible substance abuse that may be that may trigger or worsen the COFP and/or their insomnia. Insomnia treatment should begin with non-pharmacological therapy, to minimize potential side effects, drug interactions, and risk of substance abuse associated with pharmacological therapy. Behavioral therapies for insomnia include the following: sleep hygiene, cognitive behavioral therapy for insomnia, multicomponent behavioral therapy or brief behavioral therapy for insomnia, relaxation strategies, stimulus control, and sleep restriction. Approved U.S. Food and Drug Administration medications to treat insomnia include the following: benzodiazepines (estazolam, flurazepam, temazepam, triazolam, and quazepam), non-benzodiazepine hypnotics (eszopiclone, zaleplon, zolpidem), the melatonin receptor agonist ramelteon, the antidepressant doxepin, and the orexin receptor antagonist suvorexant. Chronic orofacial pain can greatly improve following treatment of the underlying insomnia, and therefore, re-evaluation of COFP is advised after 1 month of treatment.

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Cognitive-behavioral issues in the treatment and management of chronic daily headache

Cited by 22 publications

References 49 publications

Psychiatric Comorbidity in Transformed Migraine: Presentation, Treatment, Impact and Outcome

Psychiatric Comorbidity in Transformed Migraine: Presentation, Treatment, Impact and Outcome

Pain and tension-type headache: a review of the possible pathophysiological mechanisms

An update of management of insomnia in patients with chronic orofacial pain

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