Coffee Consumption and Risk of Type 2 Diabetes and Heart Disease

Campos, Hannia; Baylin, Ana

doi:10.1301/nr.2007.apr.173-179

Cited by 3 publications

(3 citation statements)

References 14 publications

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“…Coffee consumption also appeared to protect against T2DM in a dose–response manner. These findings are consistent with most studies carried out in Caucasians [26–28] and carry an important public health message. According to International Diabetes Federation’s estimations, future new cases of T2DM will predominately come from developing countries, especially China.…”

Section: Discussionsupporting

confidence: 92%

Coffee consumption is inversely associated with type 2 diabetes in Chinese

Lin

Pi-Sunyer

Chen

et al. 2011

Eur J Clin Investigation

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Section: Discussionsupporting

confidence: 92%

Coffee consumption is inversely associated with type 2 diabetes in Chinese

Lin

Pi-Sunyer

Chen

et al. 2011

Eur J Clin Investigation

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“…Coffee is one of the newer, but not very recent beverages consumed by humans, with an origin around the 9th–10th century (9). It has since become widely researched with some apparent health effects, although the evidence for this is often contradictory (54,55), with the effects possibly mediated by genetic factors (56). The evidence for coffee on inflammatory processes is conflicting, with some studies showing an anti‐inflammatory effect in some groups (57–59), but others showing no effect (46,60).…”

Section: Resultsmentioning

confidence: 99%

Inflammatory effects of nutritional stimuli: further support for the need for a big picture approach to tackling obesity and chronic disease

Egger

Dixon

2010

Obesity Reviews

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The discovery of a form of low-grade systemic inflammation (called 'metaflammation'), and the close evolutionary link between the immune and metabolic systems, poses questions about the supposed antigens (inducers) of such an immune reaction. Initially, this was thought to be mediated through obesity. However, we have identified a number of lifestyle or environmentally related inducers that may cause metaflammation, even in the absence of obesity. In this paper, the third of a series linking obesity with broad environmental and evolutionary factors, we identify nutritional stimuli with evidence of an involvement in metaflammation. From this we propose that components of certain foods and beverages with which humans have not evolved, are more often the inducers of an inflammatory effect in the body than those with which humans have become more familiar, and to which a neutral, or anti-inflammatory response may be expected to have developed. The implications of such a finding are considered in relation to broader aspects of the environment, economic growth, policy change and current global financial issues.

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“…Although some people avoid drinking coffee due to the health risks it poses, several recent studies have shown that coffee reduces the risks for developing Alzheimer's disease, 16) type 2 diabetes, 17) gallstones, 18) and colon cancer 19) and improves cognitive performance. 20,21) Recently, we reported that coffee or trigonelline, a main active component of coffee extract, prevents hearing impairment with hearing threshold shifts and delayed latencies resulting from diabetic auditory neuropathy.…”

mentioning

confidence: 99%

High-Dosage Pyridoxine-Induced Auditory Neuropathy and Protection with Coffee in Mice

Hong

Kim

et al. 2009

Biological & Pharmaceutical Bulletin

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Auditory neuropathy (AN) is a hearing disorder characterized by an abnormal auditory brainstem response (ABR). This study examined experimental AN model induced in mice following increased dosages of pyridoxine. Induced AN was examined for < or =10 weeks following the last pyridoxine treatment. To assess AN, we evaluated the ABR, auditory middle latency response (AMLR), otoacoustic emission (OAE), and histochemical morphology of the auditory nerve. Pyridoxine-treated mice exhibited an increase in the hearing threshold shift and delayed latency of both ABR and AMLR in proportion to pyridoxine dosage. Additionally, the extent of auditory nerve fiber loss increased in a dose-dependent manner following pyridoxine intoxication. Coffee or trigonelline treatment ameliorated the hearing threshold shift, delayed latency of the auditory evoked potential, and improved sensory fiber loss induced by pyridoxine intoxication. The present findings demonstrate that high-dose pyridoxine administration can be used to produce a new mouse model for AN, and coffee or trigonelline as a main active compound of coffee extract can potentially facilitate recovery from pyridoxine-induced auditory neuropathy.

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Coffee Consumption and Risk of Type 2 Diabetes and Heart Disease

Cited by 3 publications

References 14 publications

Coffee consumption is inversely associated with type 2 diabetes in Chinese

Coffee consumption is inversely associated with type 2 diabetes in Chinese

Inflammatory effects of nutritional stimuli: further support for the need for a big picture approach to tackling obesity and chronic disease

High-Dosage Pyridoxine-Induced Auditory Neuropathy and Protection with Coffee in Mice

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