Clinical Case Study

Abstract

Hence, the published formulas are not reliable in this setting. We postulate that this is due to the specific nature of LpX. Further supporting this idea is that hypercholes-terolemia on its own, i.e., without hypertriglyceridemia, is a very rare cause of significant hyponatremia; all reported cases thus far have involved the presence of LpX (9). In summary, we present a case of a 43-year-old woman with a history of PBC who was admitted for hyponatremia. Laboratory results also showed hypokale-mia and hypochloremia, but no acute kidney injury. She was found to have an extremely high TC concentration, which was verified by repeat testing. Subsequent lipopro-tein analysis detected LpX. Using direct ISE measurement , we were able to show that the patient's sodium concentration, as well as her potassium and chloride concentrations , were within normal range. This case is important owing to the degree of hypercholesterolemia, lack of lipemic sample appearance, and the link to multiple false electrolyte abnormalities.

Posterior fossa syndrome in children following tumor resection: Knowledge update

“…One of the hallmarks of CMS is that long tract signs are not present; although hemiparesis has been described in the setting of CMS, the gradual return of motor function is typically accompanied by severe ataxia, suggesting that the paresis was, in fact, a result of cerebellar dysfunction. [ 15 ] Signs of brainstem dysfunction may be present, most commonly dysarthria,[ 28 ] dysphagia,[ 20 ] and abducens or facial nerve palsies. Mood lability or so-called “pseudobulbar affect,”[ 12 20 28 ] cognitive deficits,[ 28 37 ] and urinary incontinence or retention[ 33 ] may also be present.…”