Choice of Method for Administration of Oxygen

Green, I. D.

doi:10.1136/bmj.3.5565.593

Cited by 35 publications

(15 citation statements)

References 4 publications

(4 reference statements)

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“…After the patient breathed 24% to 31% oxygen for 60 minutes either by a Ventimask (14) or nasal catheter (15), these measurements were repeated; in three patients (J. C, E. P., and F. E.) the concentration was increased from 24% to 28% after 30 minutes.…”

mentioning

confidence: 99%

Factors Contributing to the Reversible Pulmonary Hypertension of Patients with Acute Respiratory Failure Studied by Serial Observations during Recovery

Abraham

Cole²,

Green

et al. 1969

Circulation Research

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129

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Eight patients with acute respiratory failure secondary to chronic bronchitis were studied for up to 5 consecutive days following admission; cardiac output and intravascular pressures, blood volume, arterial blood gas tensions, and body weight were measured. These observations were also compared with further measurements made some weeks later just before the patient was discharged. The effects of oxygen and acetylcholine on the pulmonary circulation were also studied. Pulmonary arterial pressure was raised in all patients during their acute illness and had fallen substantially after recovery. The pulmonary arterial pressure throughout the study correlated directly with the arterial carbon dioxide and inversely with the arterial oxygen tensions. The inhalation of 24% and 28% oxygen and the infusion of acetylcholine into the pulmonary artery resulted in a fall in pulmonary arterial pressure, often to levels close to those subsequently seen after recovery from the acute illness. No significant change in cardiac output was observed. It is suggested that the acute pulmonary hypertension seen in these patients is due primarily to pulmonary vasoconstriction resulting from hypoxia. ADDITIONAL KEY WORDS hypoxic pulmonary vasoconstriction chronic bronchitis acetylcholine oxygen• Cournand and his colleagues have demonstrated that patients with acute respiratory and congestive failure secondary to chronic bronchitis and emphysema have, in general, a higher pulmonary arterial pressure than similar patients who are not in congestive failure (1). This pulmonary hypertension is associated with a lower oxygen saturation and an increased carbon dioxide tension in arterial blood. However, when these patients recover from their acute illness, the pulmo-

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confidence: 99%

Factors Contributing to the Reversible Pulmonary Hypertension of Patients with Acute Respiratory Failure Studied by Serial Observations during Recovery

Abraham

Cole²,

Green

et al. 1969

Circulation Research

Self Cite

129

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“…Der arterielle Sauerstoffdruck ist auf 67,9 ± 11,5 Torr ernied rigt, der pC02 ist geringfügig auf 43, 8 Der zusätzliche Einfluss von Atemminutenvolumen und Atemtiefe während Luftatmung und Applikation von Sauerstoff mittels Nasen sonde ist in den beiden Abbildungen 1 und 2 für einen Lungengesunden Insofern wird die Anwendung unsicher und unzureichend, worauf besonders Green [9] sowie F oitzik und Lawin [8] hingewiesen haben. Gerade für Intensiv behandlungsstationen ist das ein gewichtiger Nachteil.…”

Section: Patienten Mit Chronischen Bronchopulmonalen Erkrankungenunclassified

Zur Wirksamkeit der Sauerstoffzufuhr mittels Nasensonde

Fabel¹,

Kleine²,

Wettengel³

1971

Respiration

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4 different methods of nasal oxygen insufflation were studied in healthy adults and patients with broncho-pulmonary diseases: (1) O₂-delivery by short double nasal catheter; (2) O₂-delivery by short single nasal catheter; (3) O₂-delivery by long nasopharyngeal catheter; (4) O₂-delivery by short nasal catheter with compress (method of Poulsen). Results. In healthy adults and in hypoxemic patients the methods 3 and 4 yielded significantly higher values of arterial pO₂ (application of 2 and 41 O₂/min than the methods 1 and 2. Method 4, however, showed optimal oxygen delivery in combination with optimal humidification and warming of gas and should be prefe-red. A paradoxical fall of arterial pO2 during nasal oxygen administration is caused by augmented tidal and minute volume. This phenomena is demonstrated and discussed.

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“…1 Generally, an oxygen flow of 5 L/min can provide an F IO 2 of no more than 0.50. [2][3][4] Besides oxygen flow, the F IO 2 depends on several other factors, such as breathing pattern and volume of the oral cavity as an oxygen reservoir. Additionally, sedative drugs have been shown to induce changes in the breathing pattern.…”

Section: Introductionmentioning

confidence: 99%

“…5 Benzodiazepines and propofol cause low-V T and high-breathing frequency patterns. 6,7 Although some reports have measured the F IO 2 during oxygen delivery via a nasal cannula, 2,4,8 the influence of breathing pattern on the F IO 2 has not been investigated.…”

Section: Introductionmentioning

confidence: 99%