Childhood Cutaneous Lymphoid Hyperplasia Following Feline Scratches

Madhogaria, Sharmila; Carr, Richard; Gach, J. E.

doi:10.1111/j.1525-1470.2010.01142.x

Cited by 6 publications

(5 citation statements)

References 16 publications

(16 reference statements)

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“…On the other hand, 80% of patients revealed intensive pandermal infiltration, predominantly with CD4-positive T cells, in drug-related lesions. When reviewing previous case reports that analyzed clinicopathological features according to the causative agents mentioned above, some were comparable to our results but others showed somewhat different manifestations [1,[15][16][17]. Moreover, a variety of pathological features can be manifested according to specific agents in the same group.…”

Section: Discussionsupporting

confidence: 88%

Clinical and Histopathological Characteristics of Cutaneous Lymphoid Hyperplasia: A Comparative Study According to Causative Factors

Choi

Lee

Lim

et al. 2020

JCM

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Cutaneous lymphoid hyperplasia (CLH) is a heterogeneous type of reactive lymphocytic infiltration resembling cutaneous lymphoma clinically and histopathologically. Few studies describe the relationship between the causative agents and histopathological and immunohistochemical characteristics of CLH. We investigated the clinical and histopathological characteristics of 50 patients with cutaneous CLH and analyzed them according to causative factors and predominant cell types (B or T cells). We retrospectively reviewed medical records to identify causative agents, and histopathological and immunohistochemical features. The majority of infiltrating lymphocytes were T cells (60%). T cell-dominant CLH showed papuloplaque lesions, whereas B cell-dominant CLH lesions were nodular. The infiltration pattern differed between T and B cells. In terms of prognosis, B-cell-predominant lesions tended to respond better to treatment than T-cell-predominant lesions. Hair dyes tended to be associated with multiple CLH lesions in older patients. CLH lesions associated with drugs were located on the head and neck. Insect bites were likely to cause a solitary papular lesion. Histopathologically, infiltration depth was located more superficially than other causes and featured intense eosinophilic infiltration. Thus, our study demonstrated that CLH presents different clinicopathological features according to causative agents and predominant lymphocytic types.

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Section: Discussionsupporting

confidence: 88%

Clinical and Histopathological Characteristics of Cutaneous Lymphoid Hyperplasia: A Comparative Study According to Causative Factors

Choi

Lee

Lim

et al. 2020

JCM

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“…Another case in a child suggested cutaneous lymphoid hyperplasia secondary to recurrent inoculation of Bartonella henselae due to feline scratches. The lesions responded to azithromycin and disappeared completely after the cat died (40).…”

Section: Treatment According To Cutaneous Pseudolymphoma Aetiologymentioning

confidence: 98%

Treatment of Cutaneous Pseudolymphoma: A Systematic Review

Miguel¹,

Peckruhn²,

Elsner³

2018

Acta Derm Venerol

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Cutaneous pseudolymphoma (CPL) is a reactive polyclonal T- or B-cell lymphoproliferative process. CPL may appear as localized or disseminated skin lesions. While most cases of CPL are idiopathic, they may also occur as a response to, for example, contact dermatitis, arthropod reactions, and bacterial infections. CPL can be classified based on its clinical features, but all variants have similar histopathological patterns of either predominantly B-cell infiltrates, T-cell infiltrates, or mixed T/B-cell infiltrates. The prognosis of CPL is good, but the underlying disease process should be taken into account. If an antigenic stimulus is identified, it should be removed. In patients with idiopathic CPL, a close follow-up control strategy should be adopted. The aim of this systematic review is to summarize all reported treatments for CPL. The review was based on articles from the PubMed database, using the query "skin pseudolymphoma treatment", English and German, about "human" subjects, and published between 1990 and 2015 documenting adequate treatment and/or aetiology. Mainly individual case reports and small case series were found. Treatment options include topical and intralesional agents, systemic agents, and physical modalities. The final part of the review proposes a treatment algorithm for CPL according to each aetiology, based on the literature of the last 25 years. Future research should focus on randomized controlled trials and studies on long-term outcomes, which were not identified in the current review.

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“…La etiopatogenia de estas lesiones es desconocida, pudiendo en algunos casos ocurrir como una respuesta inmunológica local reactiva a desencadenates tales como, picadura de insectos, acupuntura, vacunas, tatuajes, fármacos, exposición a alérgenos de contacto o asociado a infecciones por herpes zóster, Borrelia burgdorferi, Leishmania donovani o molusco contagioso. Los fármacos más frecuentemente asociados a estas lesiones son fenitoína, inhibidores de la enzima convertidora de la angiotensina II, antihistamínicos y menos frecuentemente bromocriptina y cefuroxima Gambichler et al;Gissler & Heininger;Nihal et al;Madhogaria et al;Stavrianeas et al, 2007;Magro & Crowson, 1996;Saeed et al, 2000). En ambas pacientes el uso de losartan (inhibidor de la enzima convertidora de la angiotensina II) y fenitoína son el único factor desencadenante asociado.…”

Section: Discussionunclassified

“…El curso de esta enfermedad es benigno, desapareciendo las lesiones espontáneamente en semanas o meses, pudiendo, en algunos casos persistir en forma indefinida y/o recidivar; existiendo incluso reportes aislados de progresión a linfoma. Dada la posibilidad de progresión maligna, es que algunos autores plantean la interrogante en cuanto a la naturaleza de estos tumores, postulando que los pseudolinfomas representarían mas bien al espectro benigno en la escala evolutiva hacia un proceso linfoproliferativo maligno Gissler & Heininger;Madhogaria et al;Ploysangam et al, 1998;Bouloc et al, 1999).…”

Section: Discussionunclassified

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Hiperplasia Cutánea Linfoide (Linfocitoma cutis) Secundaria a Fármacos: Hallazgos Clínicos y Morfológicos

Valenzuela

2012

Int. J. Morphol.

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Childhood Cutaneous Lymphoid Hyperplasia Following Feline Scratches

Cited by 6 publications

References 16 publications

Clinical and Histopathological Characteristics of Cutaneous Lymphoid Hyperplasia: A Comparative Study According to Causative Factors

Clinical and Histopathological Characteristics of Cutaneous Lymphoid Hyperplasia: A Comparative Study According to Causative Factors

Treatment of Cutaneous Pseudolymphoma: A Systematic Review

Hiperplasia Cutánea Linfoide (Linfocitoma cutis) Secundaria a Fármacos: Hallazgos Clínicos y Morfológicos

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