Changes of Tc-99m HMPAO Brain Distribution in Herpes Encephalitis

Scao, Y. Le; Turzo, A.; M, Guias; Menn, G. Le; Garre, Ml; Morin, Pascale

doi:10.1097/00003072-199305000-00023

Cited by 8 publications

(3 citation statements)

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“…Herpes simplex encephalitis causes a focal brain destruction mainly located to the temporal lobes. The encephalitis is initially associated with a focal hyperaemia which at later stages reverts to a hypoaemia (69)(70)(71).…”

Section: Infectious Diseasesmentioning

confidence: 99%

SPECT measurements of brain function in dementia; a review

Ryding

1996

Acta Neurologica Scandinavica

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A brief review of the role of SPECT (single photon emission computed tomography) in the diagnosis of dementing brain disease is given. Between 1990 and 1995 some 100 original articles deal with SPECT measurements with radioactive tracers of brain function in demented patients. The main field is measurement of regional cerebral blood flow (rCBF) but there is an increasing number of studies concerning receptor functions in the demented brain.

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Section: Infectious Diseasesmentioning

confidence: 99%

SPECT measurements of brain function in dementia; a review

Ryding

1996

Acta Neurologica Scandinavica

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“…Some studies of herpes encephalitis have shown hyperperfusion in the inflammatory tissue using 99m Tc HMPAO-single photon emission tomography (SPECT) and PET [4][5][6][7]. MR perfusion imaging following a bolus injection of Gd-DTPA does also allow imaging of different perfusion parameters in brain tissue.…”

Section: Introductionmentioning

confidence: 99%

MR Perfusion Imaging in Acute Herpes Simplex Encephalitis

Stracke¹,

Meister²,

Möller-Hartmann³

et al. 2006

Clin Neuroradiol

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“…The course that hypermetabolism by PET in subclinical ictal phase progressed to hypoperfusion by SPECT may also suggest the existence of epileptic event. 6 Crossed cerebellar hypermetabolism is also a striking feature. Transtentorial diaschisis is not seen in patients with Creutzfeldt-Jakob disease using I-123 HIPDM brain SPECT.…”

mentioning

confidence: 99%

Cerebral Hypermetabolism Demonstrated by FDG PET in Familial Creutzfeldt-Jakob Disease

Nagasaka¹,

Nagasaka²,

Ohta³

et al. 2011

Clinical Nuclear Medicine

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Right cerebral and contralateral cerebellar hypermetabolism were observed on FDG PET in a 68-year-old woman with familial Creutzfeldt-Jakob disease (CJD) at an early stage before seizures occurred. The disease progressed with frequent seizures, myoclonus, and a startle reaction. In all past reports, FDG PET studies demonstrated hypometabolism in the cerebrum, cerebellum, and thalamus in patients with CJD. Focal hypermetabolism corresponding with epileptic foci is a common finding in ictal epilepsy patients, and hypometabolism is common in patients with myoclonus or the startle reaction. This finding may reflect a prodromal pathophysiology of epilepsy. Attention should be paid to the diagnosis of CJD while using FDG PET.

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Changes of Tc-99m HMPAO Brain Distribution in Herpes Encephalitis

Cited by 8 publications

References 0 publications

SPECT measurements of brain function in dementia; a review

SPECT measurements of brain function in dementia; a review

MR Perfusion Imaging in Acute Herpes Simplex Encephalitis

Cerebral Hypermetabolism Demonstrated by FDG PET in Familial Creutzfeldt-Jakob Disease

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