Abstract:Prenatal exposure to N-cholinoblocker gangleron and in lesser degree to M-cholinoblocker metamisil leads to significant violations of the motivational component of sexual function in sexually mature offspring of males, expressed in low values of primary sexual activity, and increased latency of the approach and sexual dysfunction after the acquisition of sexual experience. Among the causes of sexual dysfunction in the offspring, it can be noted a change in the hormonal background in male rats, in the form of a… Show more
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