Changes Caused by Methidathion in Activity and Distribution of Molecular Forms of Carp (Cyprinus carpio L.) AChE

Effects of Carbofuran, Diuron, and Nicosulfuron on Acetylcholinesterase Activity in Goldfish (Carassius auratus)

“…This observation con"rms preliminary results on zebra"sh, Brachydanio rerio (Bertrand et al, 1998), and is compatible with earlier results of Guillon and MassoulieH (1976), who found three molecular forms in gold"sh (18S, 12S, and 5S). In contrast, recent reports on carp muscles revealed a di!erent pattern of molecular forms with a minority of A12 form (15.5%) and a majority of G4/A4 form (84.5%) (Szegletes et al, 1995). It is known that AChE molecular forms are susceptible to autolytic degradation and that A forms are readily converted into their G4 constituents, especially if extraction bu!ers lack antiproteolytics (Silman et al, 1978).…”

“…This form was not found in the embryos exposed to the lowest concentrations of carbofuran (2, 10 g/L) and might be a sign of AChE neosynthesis after this massive inhibition. In common carp, Szegletes et al have found that the proportions of muscle molecular forms in response to a 120-h exposure to methidathion, an organophosphate, caused a slight decrease of A12 in skeletal and heart muscles and appearance of the G1 form in both tissues (Szegletes et al, 1995).…”

Mixture Toxicity of Bensulfuron-Methyl and Acetochlor to Red Swamp Crayfish (Procambarus clarkii): Behavioral, Morphological and Histological Effects

“…This may explain the lesser histological effects of MBA on muscles in this study. Several other investigators have also detected the lowest concentrations of toxicants in fish and crayfish muscles [ 52 , 53 , 54 ]. In addition, the hard carapace of crayfish may absorb a portion of toxicants and reduce the amount of toxicants entering muscles from the body surface.…”

Recovery of Acetylcholinesterase Activity in the Common Carp ( Cyprinus carpio L.) After Inhibition by Organophosphate and Carbamate Compounds

“…We study teleostean brain AChE, particularly in common carp, because of its great geographical distribution throughout the world (Billard 1995). Previous investigators have characterised AChE in common carp using different extraction techniques and inhibitors (Szabo et al 1992;Szegletes et al 1995). The objectives of this paper were: firstly to extend the observations of Szabo et al (1992) by investigating the common carp brain AChE, secondly to determine the type of inhibition induced by carbofuran (C) in chlorfenvinphos (OP) on the common carp brain AChE, and finally to determine the in vivo required time for the recovery of brain AChE activity of the common carp after inhibition by these two insecticides.…”