1988
DOI: 10.1055/s-2007-1020082 View full text |Buy / Rent full text
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Abstract: An increase of arterial carbon dioxide (CO2) partial pressure induces an increase of cerebral blood flow by dilatation of the resistance vessels. By the Transcranial Doppler sonographic technique (TCD) blood flow velocity as a correlate of flow volume can be measured within the great basal intracranial arteries. We investigated 8 patients with an internal carotid artery occlusion or high-grade stenosis and 5 cerebrovascular diseased patients without extracranial stenosis. 12 healthy volunteers and patients wit… Show more

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“…[6][7][8] An ipsilaterally lowered CO 2 reactivity can be found in patients with severe stenosis of the ICA, especially when the contralateral ICA is also obstructed. [9][10][11][12][13][14] The CO 2 reactivity might be superior to the measured degree of ICA stenosis alone as an indicator of the hemodynamic consequences of ICA stenosis, because CO 2 reactivity also depends on the quality of the collateral circulation. As illustration of the clinical significance of the CO 2 reactivity, especially in patients with low-flow infarctions, patients with chronic retinal ischemia or hypostatic TIAs have a lowered CO 2 reactivity.…”
Section: Discussionmentioning
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“…[6][7][8] An ipsilaterally lowered CO 2 reactivity can be found in patients with severe stenosis of the ICA, especially when the contralateral ICA is also obstructed. [9][10][11][12][13][14] The CO 2 reactivity might be superior to the measured degree of ICA stenosis alone as an indicator of the hemodynamic consequences of ICA stenosis, because CO 2 reactivity also depends on the quality of the collateral circulation. As illustration of the clinical significance of the CO 2 reactivity, especially in patients with low-flow infarctions, patients with chronic retinal ischemia or hypostatic TIAs have a lowered CO 2 reactivity.…”
Section: Discussionmentioning
“…In CTO of the ICA, CVR is reduced; this impairment has been associated with increased risk of ischemic events and may be useful for stroke risk stratification ( 30 ). In normal cases, CVR is unimpaired (stage 0) ( 31 ). CVR impairment may be divided into three stages: In stage I hemodynamic failure, autoregulatory compensatory vasodilation is still able to maintain normal CBF, and the oxygen extraction fraction (OEF) remains normal.…”
Section: Hemodynamicsmentioning