Central diabetes insipidus following intraventricular hemorrhage

Adams, James M.; Kenny, John D.; Rudolph, Arnold J.

doi:10.1016/s0022-3476(76)81003-7

Cited by 23 publications

(3 citation statements)

References 7 publications

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“…Severe brain damage due to birth trauma or asphyxia, intraventricular hemorrhage, group B streptococcal meningitis and congenital brain anomaly [9][10][11][12] are among the reported causes in this age group. The diagnosis of DI may be delayed as the classic features of polyuria and polydipsia may not be obvious at this young age and are replaced by nonspecific symptoms such as irritability and failure to thrive.…”

Section: Discussionmentioning

confidence: 99%

Central Diabetes Insipidus Complicating Congenital Toxoplasmosis: A Case Report

Benjamin

Al-Amari

1995

Annals of Saudi Medicine

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Section: Discussionmentioning

confidence: 99%

Central Diabetes Insipidus Complicating Congenital Toxoplasmosis: A Case Report

Benjamin

Al-Amari

1995

Annals of Saudi Medicine

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“…Central DI in the neonate has been reported following meningitis [9] and intraventricular hemorrhage [10]. Transient DI has been related to disseminated intravascular coagulation (DIC) in an infant who had listeriosis [23].…”

Section: Central Diabetes Insipidusmentioning

confidence: 99%

Empty Sella Syndrome, Panhypopituitarism, and Diabetes Insipidus

Jones

Drut

Valencia

et al. 2005

Fetal and Pediatric Pathology

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We present an 18-month-old girl with short stature, obesity, panhypopituitarism, diabetes insipidus, and visual defects. Postmortem examination revealed brain atrophy due to a diffuse encephalopathy, numerous calcified neurons in cerebral cortex, deep telencephalic and diencephalic nuclei, diffuse neuronal necrosis in hypothalamic nuclei, moderate atrophy of optic nerves, very thin hypophyseal stalk, and empty sella with the hypophysis compressed to the dorsal aspect of the concavity. Our hypothesis is that the presence of an empty sella in a child with hypophyseal-hypothalamic abnormalities should alert physicians to the existence of hypothalamic lesions secondary to a perinatal insult. We discuss the possible pathogenesis of these findings as well as lines of evidence available in the literature.

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“…A VP Release Recent studies on the pattern of AVP secretion in the immediate neonatal period have shown higher cord plasma AVP con centrations in newborn infants born vaginally than in those delivered by cesarean sec tion [9] and perinatal complication such as asphyxia [27], hypoxic-ischemic encephalop athy [12], intracranial hemorrhage [1], se vere respiratory distress [38], pneumotho rax, pneumonia, atelectasis [20,30], and meningitis [19] have been reported to be associated with elevated plasma and urinary AVP concentrations. The inappropriate se cretion of AVP related to perinatal pathol ogy is thought to be an important cause of the early type of hyponatremia which fre quently occurs in the first week of life [18].…”

Section: Observations In Human Neonatesmentioning

confidence: 99%

Renal Response to Vasopressin in Premature Infants: What Is New?

Sulyok¹

1988

Neonatology

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Clinical and experimental data indicate that the neonatal pituitary is capable of responding with arginine vasopressin (AVP) release to physiological stimulation and pathological events commonly seen in the perinatal period. The limited concentrating performance of the newborn kidney is thought, therefore, to be accounted for by the diminished end-organ responsiveness to AVP and the inability of the immature kidney to generate and maintain deep corticopapillary osmotic gradient. Evidence has been provided to indicate that in low birth weight premature infants the impaired renal tubular sodium transport, the renal salt wasting and late hyponatremia, and the increased renal prostaglandin production may be important factors in attenuating renal response to AVP.

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Central diabetes insipidus following intraventricular hemorrhage

Cited by 23 publications

References 7 publications

Central Diabetes Insipidus Complicating Congenital Toxoplasmosis: A Case Report

Central Diabetes Insipidus Complicating Congenital Toxoplasmosis: A Case Report

Empty Sella Syndrome, Panhypopituitarism, and Diabetes Insipidus

Renal Response to Vasopressin in Premature Infants: What Is New?

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