Cardiac troponin I release in non-ischemic reversible myocardial injury from parvovirus B19 myocarditis

Baig, Muhammad A.; Ali, Shaukat; Khan, Mutahir U.; Rasheed, Javeria; Qadir, Abdul; Vasavada, Balendu C.; Khan, Ijaz A.

doi:10.1016/j.ijcard.2006.06.048

Cited by 15 publications

(10 citation statements)

References 13 publications

(9 reference statements)

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“…Secondary ischemic cardiac injury describes myocardial ischemia with myocyte injury in the absence of atherosclerotic plaque rupture and due to increased myocardial oxygen demand that outstrips myocardial oxygen supply. Nonischemic cardiac injury describes cTn release caused by direct damage to the myocardium, including blunt trauma (24), penetrating trauma (25), myocarditis (26), or drug and toxin-induced cardiotoxicity (27). At the present time, the release of cTn by healthy individuals after exercise cannot be explained by any of these pathophysiologic scenarios.…”

Section: Troponin Biochemistry and Routine Clinical Usementioning

confidence: 99%

Exercise-Induced Cardiac Troponin Elevation

Shave

Baggish

George

et al. 2010

Journal of the American College of Cardiology

355

138

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Regular physical exercise is recommended for the primary prevention of cardiovascular disease. Although the high prevalence of physical inactivity remains a formidable public health issue, participation in exercise programs and recreational sporting events, such as marathons and triathlons, is on the rise. Although regular exercise training reduces cardiovascular disease risk, recent studies have documented elevations in cardiac troponin (cTn) consistent with cardiac damage after bouts of exercise in apparently healthy individuals. At present, the prevalence, mechanism(s), and clinical significance of exercise-induced cTn release remains incompletely understood. This paper will review the biochemistry, prevalence, potential mechanisms, and management of patients with exercise-induced cTn elevations.

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Section: Troponin Biochemistry and Routine Clinical Usementioning

confidence: 99%

Exercise-Induced Cardiac Troponin Elevation

Shave

Baggish

George

et al. 2010

Journal of the American College of Cardiology

355

138

View full text Add to dashboard Cite

show abstract

“…In the present case PVB19 induced perimyocarditis without cardiac dysfunction, progressing to a situation where the cardiac function severely deteriorated associated with the release of cardiac enzymes, as caused by reversible and/or irreversible non-ischemic myocardial injury [3]. This rapid deterioration was caused by GCM.…”

Section: Discussionmentioning

confidence: 54%

Giant cell myocarditis triggered by a parvovirus B19 infection

Dennert¹,

Schalla²,

Suylen³

et al. 2009

International Journal of Cardiology

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“…14 Although the present case presented with heart failure and dyspnoea, cases may present with chest pain, malaise, oedema or elevation of cardiac enzymes such as creatine kinase or troponin I. 15 Clinical course of parvovirus myocarditis is variable ranging from full recovery to heart failure and death. Failure to eliminate the virus from myocardium is associated with adverse prognosis.…”

Section: Discussionmentioning

confidence: 79%