Cardiac Arrest caused by Barlow's Syndrome or by Stress Cardiomyopathy?

Facciorusso, Antonio; Amico, Cesare; Vigna, Carlo; Santoro, Tiberio; Potenza, Domenico; Massaro, Rosalba; Stanislao, Mario; Valle, Guido; Cavaliere, Lucio; Fanelli, Raffaele

doi:10.1016/j.ijcard.2008.11.071

Cited by 8 publications

(6 citation statements)

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“…At admission the patients complained of chest pain (n: 16, 80%), suffered from syncope (n: 2, 10%) or cardiac arrest [11] (n: 2, 10%). All TTC patients presented initial ECG consistent findings with ACS (ST-segment elevation).…”

Section: Resultsmentioning

confidence: 99%

“…Moreover, provocative tests with ergonavine or acetylcholine induced multivessel spasm only in 28% of TTC patient 16 . Other theories focus on catecholamines: in fact, abnormally high levels of serum catecholamines have been recorded in TTC patients [11,17]. In an animal model, the prolonged immobilization induced TTC through the increase of catecholamines and the up-regulation of heart adrenoreceptors [18].…”

Section: According To Literaturementioning

confidence: 99%

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2017

BAOJPM

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Section: Resultsmentioning

confidence: 99%

Section: According To Literaturementioning

confidence: 99%

Untitled

2017

BAOJPM

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“…Takotsubo cardiomyopathy has been associated with an increase in plasma catecholamines, like epinephrine, norepinephrine, and dopamine. 7 , 8 As per the Mayo criteria for cardiomyopathy all four of the following criteria should be present. Criterion 1: Transient hypokinesis, akinesis, or dyskinesis of left ventricular segments and may or may not involve apical wall.…”

Section: Discussionmentioning

confidence: 99%

An unusual case report of stress-induced cardiomyopathy presenting as ventricular fibrillation cardiopulmonary arrest and third-degree atrioventricular block

Parekh

Sengupta

Zainea

2021

European Heart Journal - Case Reports

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Background Takotsubo cardiomyopathy (TCM) can clinically present as an acute coronary syndrome; however, the former has regional wall motion abnormalities that extend beyond a single coronary vascular territory without any plaque rupture. Takotsubo cardiomyopathy classically involves apical ballooning of left ventricle (LV). It is uncommon for TCM to present as cardiopulmonary arrest (CPA) along with third-degree atrioventricular (AV) block. Case summary A 63-year-old female, underwent a ventricular fibrillation (VF) CPA. She was defibrillated three times and return of spontaneous circulation (ROSC) was achieved after 37 min. Her post-ROSC electrocardiogram showed non-specific ST-segment changes and T-wave inversions and soon progressed to third-degree AV block. Patient had a transvenous pacemaker placed to pace her heart. Echocardiogram showed an LV ejection fraction of 15–20% with akinesis of the apex and anteroseptum. An echocardiogram repeated 4 days after the cardiopulmonary arrests showed an ejection fraction of 60–65% with hypokinesis of mid anterior and antero-apical hypokinesis. However, the patient still continued to require a pacemaker and hence eventually received a dual-chamber pacemaker/implantable cardioverter-defibrillator for her AV block and ventricular arrhythmia. Discussion Most commonly TCM presents with chest pain and symptoms of acute myocardial infarction. We present a very rare presentation of TCM associated with VF and CPA along with third-degree AV block. There have handful of case reports documenting TCM causing CPA in some patients and other case reports showing TCM causing high degree AV block. In our patient, TCM was associated with both VF and CPA along with third-degree AV block.

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“…However, if right atrial pressure increases, as in the case of pulmonary embolism or chronic obstructive pulmonary disease, a right to left shunt arises with risk for paradoxical embolism [1]. This condition is caused by a venous embolus that crosses the PFO or that is trapped in PFO [4][5][6]. Brain arteries are the most frequent localization of paradoxical embolism [4]; less commonly paradoxical embolism has been described in the upper limb [1], coronary [8], renal or retinal arteries [9].…”

Section: Discussionmentioning

confidence: 99%

“…However, if right atrial pressure increases, a right to left shunt arises with risk for paradoxical embolism. This condition is caused by a venous embolus that crosses the PFO or that is trapped in PFO [4][5][6]. Brain arteries are the most frequent localization of paradoxical embolism [4].…”

Section: Introductionmentioning

confidence: 99%