Carcinoma of the floor of the mouth

Abstract

CARCINOMA of the floor of the mouth is a clinical an epithelioma varying in degree of keratinization entity only in its early stages. Involvement of and anaplasia; it is either an exuberant mass or adjacent structures is common and soon the disease an ulcer with indurated edges. The disease is spreads beyond the floor itself. considered as in three directions-to the intrinsic patients, many tumours develop in edentulous and extrinsic muscles of the tongue, to the gum mouths. The usual so-called precancerous changes with or without involvement of the alveolus, and such as leucoplakia, chiefly of the floor itself and to the cervical lymph-glands. Although such a of the gingival mucosa, are frequently seen. All subdivision is artificial it is of help in considering these features are well known and require no the choice of treatment and in the planning thereof emphasis. (Fig. 267).

An epidemiologic study of malignant oral tumors in Sweden 1958–77

“…Firstly, many times it is only [Possible to separate early cases ol cancer in the tongue and iloor of mouth as clinical anatomic entities. In most cases of advanced tumor stages, adjacent tissue structures are involved ineluding extension to the gingiva, alveolar mueosa, soft palate and pharyngeal wall (22,28,29). Seeondly, one has to consider that the anatomic subclassifications used in different countries vary to some extent.…”

Oral metal contact allergy: a pilot study on the cause of oral squamous cell carcinoma

“…Well‐known risk factors for the genesis of intraoral cancer include alcohol, tobacco, or betel nut use 1 . The roles of genetic influence, 2–8 exposure to human papillomavirus, 1,9,10 and a background of oral lichen planus (OLP) 11–13 continue to be explored. Contact allergy has been mentioned only briefly as having a role in the genesis of oral cancer 14 …”

Oral Metal Contact Allergy: A Cause of Oral Squamous Cell Carcinoma? A Pilot Study

“…Well-known risk factors for the genesis of intraoral cancer include alcohol, tobacco, or betel nut use. 1 The roles of genetic influence, [2][3][4][5][6][7][8] exposure to human papillomavirus, 1,9,10 and a background of oral lichen planus (OLP) [11][12][13] continue to be explored. Contact allergy has been mentioned only briefly as having a role in the genesis of oral cancer.…”