Budd-Chiari syndrome

Wang, Zhonggao; Jones, R. Scott

doi:10.1016/s0011-3840(96)80001-3

Cited by 47 publications

(27 citation statements)

References 152 publications

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“…Another advantage of venography is that the portal vein can be assessed at the same sitting and if found to be occluded can be dilated with or without stenting in order to decompress the liver. Pressure measurements obtained during venography provide useful information before attempting surgical decompression [135,136] . Also, pressure measurements are useful in the post-surgical follow-up of these patients.…”

Section: Hepatic Venographymentioning

confidence: 99%

“…Mesoatrial shunt has a high rate of thrombosis due to relatively low flow rate in the prosthetic graft [126] . Side-to-side portocaval shunt and cavoatrial shunt have been described for patients with both caval and hepatic venous thrombosis [136,157,158] . In addition to shunts, other surgical procedures can also provide adequate decompression in selected patients.…”

Section: Managementmentioning

confidence: 99%

“…In addition to shunts, other surgical procedures can also provide adequate decompression in selected patients. Transatrial membranotomy can be effective in patients with IVC membranes [136,157,159,160] . Dorsocranial resection of the liver is the only feasible procedure if there is both portal and superior mesenteric vein obstruction [161] .…”

Section: Managementmentioning

confidence: 99%

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Hepatic venous outflow obstruction: Three similar syndromes

Bayraktar

Seren²,

Bayraktar³

2007

WJG

116

110

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Our goal is to provide a detailed review of venoocclusive disease (VOD), Budd-Chiari syndrome (BCS), and congestive hepatopathy (CH), all of which results in hepatic venous outflow obstruction. This is the first article in which all three syndromes have been reviewed, enabling the reader to compare the characteristics of these disorders. The histological findings in VOD, BCS, and CH are almost identical: sinusoidal congestion and cell necrosis mostly in perivenular areas of hepatic acini which eventually leads to bridging fibrosis between adjacent central veins. Tender hepatomegaly with jaundice and ascites is common to all three conditions. However, the clinical presentation depends mostly on the extent and rapidity of the outflow obstruction. Although the etiology and treatment are completely different in VOD, BCS, and CH; the similarities in clinical manifestations and liver histology may suggest a common mechanism of hepatic injury and adaptation in response to increased sinusoidal pressure.

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Section: Hepatic Venographymentioning

confidence: 99%

Section: Managementmentioning

confidence: 99%

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Hepatic venous outflow obstruction: Three similar syndromes

Bayraktar

Seren²,

Bayraktar³

2007

WJG

116

110

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“…It is often thought that membranous lesions are congenital in origin; however, the mechanisms that result in their formation are not clear despite numerous epidemiological studies [16] . The interesting point in this regard is that the higher number (n = 5) of older patients with web-based BCS is not consistent with the congenital formation of web obstructions.…”

Section: Discussionmentioning

confidence: 99%

Presentation of Hospital Outcomes and Different Treatment Methods of Patients with Budd-Chiari Syndrome: A Report from Two Tertiary Hospitals in Iran

Ebrahimi

Modaghegh²,

Esmaeilzadeh³

2011

Med Princ Pract

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Objective: The aim of this study was to report common presentations of Budd-Chiari syndrome (BCS) and the early outcome of different treatment methods in two tertiary hospitals in Iran. Subjects and Methods: This case series study was performed on 21 patients (mean age: 42 ± 13.09 years; 11 male, 52.4%, and 10 female, 47.6%) admitted for treatment of BCS in two tertiary referral centers in Mashhad, Iran, between 2002 and 2008. All required data of signs, underlying etiology, treatment methods and in-hospital mortality were gathered from patients’ medical records. Results: Angiographic and sonographic findings showed that the most frequent isolated location of obstruction was the inferior vena cava (n = 12, 57.1%). No distinct underlying disease was found in 6 (28.6%) patients. Eleven (52.4%) patients had web obstruction and 4 patients had other related underlying diseases. Treatment modalities consisted of medical follow-up in 12 (57.1%), angioplasty in 6 (28.6%), and surgery in 3 (14.3%) patients. Medical follow-up of 3 patients, 1 with angioplasty and 2 who had undergone surgery, disclosed that they had died before discharge from hospital. Conclusion: Higher age at diagnosis may reflect late diagnosis at an advanced stage of disease. We suggest that the early symptoms of this disease should be taken into account more seriously in differential diagnosis. Balloon angioplasty seems to be a more efficient method for treatment of BCS.

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“…A classification suggested by prof. Wang included eight types based on anatomical location and the extent of the outflow obstruction (Wang and Jones, 1996). Less complex classifications used at present divide BCS according to the velocity of its development to fulminant, acute, subacute and chronic and by etiology as primary or secondary BCS .…”

Section: Classificationmentioning

confidence: 99%

Budd-Chiari Syndrome

et al. 2017

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Budd-Chiari syndrome (BCS) is a rare disease with an incidence of 0.1 to 10 per million inhabitants a year caused by impaired venous outflow from the liver mostly at the level of hepatic veins and inferior vena cava. Etiological factors include hypercoagulable conditions, myeloprolipherative diseases, anatomical variability of the inferior vena cava, and environmental conditions. Survival rates in treated patients range from 42 to 100% depending on the etiology and the presence of risk factors including parameters of Child-Pugh score, sodium and creatinine plasma levels, and the choice of treatment. Without treatment, 90% of patients die within 3 years, mostly due to complications of liver cirrhosis. BCS can be classified according to etiology (primary, secondary), clinical course (acute, chronic, acute or chronic lesion), and morphology (truncal, radicular, and venooclusive type). The diagnosis is established by demonstrating obstruction of the venous outflow and structural changes of the liver, portal venous system, or a secondary pathology by ultrasound, computed tomography, or magnetic resonance. Laboratory and hematological tests are an integral part

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Budd-Chiari syndrome

Cited by 47 publications

References 152 publications

Hepatic venous outflow obstruction: Three similar syndromes

Hepatic venous outflow obstruction: Three similar syndromes

Presentation of Hospital Outcomes and Different Treatment Methods of Patients with Budd-Chiari Syndrome: A Report from Two Tertiary Hospitals in Iran

Budd-Chiari Syndrome

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