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Cited by 2 publications
(2 citation statements)
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References 32 publications
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“…23 The cytoplasmic localization partially contradicts the consensus about the sole nuclear localization of BMI-1 in the neoplastic and the potentially malignant lesions showing high proliferative activity reported in previous studies. [24][25][26] The stromal fibroblasts in both groups of the present study showed strong intra-nuclear immunoreactivity to the BMI-1. This is in agreement with what was previously reported that BMI-1 has been shown to immortalize normal fibroblasts via reactivation of the human telomerase reverse transcriptase gene in these cells.…”
Section: Discussionsupporting
confidence: 54%
“…23 The cytoplasmic localization partially contradicts the consensus about the sole nuclear localization of BMI-1 in the neoplastic and the potentially malignant lesions showing high proliferative activity reported in previous studies. [24][25][26] The stromal fibroblasts in both groups of the present study showed strong intra-nuclear immunoreactivity to the BMI-1. This is in agreement with what was previously reported that BMI-1 has been shown to immortalize normal fibroblasts via reactivation of the human telomerase reverse transcriptase gene in these cells.…”
Section: Discussionsupporting
confidence: 54%
“…An increased number of proliferating cells can be observed in tissues with chronic inflammation as well as in tissues displaying hyperplasia in comparison with healthy tissues. Indices using the proliferation marker Ki-67 have been utilized in studies of LPL [ 18 , 19 ] but also OLP [ 20 , 21 ]. Because OLP and LPL present with different stages of chronic inflammation and altered epithelia, Ki-67 is a biomarker that can be used to compare cell proliferation rates in the two disorders.…”
Section: Introductionmentioning
confidence: 99%