“…Structural competency of the longitudinal arch is therefore critical in humans. Indeed, clinical work has outlined numerous risk factors associated with the surgical release of the plantar aponeurosis, including a decrease in arch height (Kim and Voloshin, 1995;Kitaoka et al, 1997;Sharkey et al, 1998) and altered stress distributions in hard (i.e., calcaneocuboid joint and metatarsals) and soft (i.e., spring ligament, short plantar ligament, long plantar ligament, flexor digitorum longus, and flexor hallucis longus) tissues (Murphy et al, 1998;Gefen, 2002;Crary et al, 2003;. A well-developed human plantar aponeurosis contributes to propulsion by rapidly transmitting forces from the hind foot to the forefoot via elastic recoil of the ''preloading'' and peakloading phases (Hicks, 1954;Wright and Rennels, 1964;Bojsen-Møller, 1979;Ker et al, 1987;Simkin and Leichter, 1990;Pataky et al, 2008;Caravaggi et al, 2009).…”