Bioconcentration of trichlorfon insecticide in pacu (Piaractus mesopotamicus)

Lopes, Ruy Bessa; Paraíba, L. C.; Ceccarelli, Paulo S.; Tornisielo, Valdemar Luiz

doi:10.1016/j.chemosphere.2005.11.029

Cited by 47 publications

(32 citation statements)

References 15 publications

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“…The renewed water contained nominal exposure concentrations to ensure the desired concentrations of trichlorfon in the water. The half-life time of trichlorfon in water was 2.5 days, and the dissipation time of 95% trichlorfon in water was 10.2 days (Lopes et al 2006). Fish were fed twice daily with commercial fish pellets.…”

Section: Experimental Designmentioning

confidence: 99%

“…Its dosage to eradicate ectoparasites varies from 0.1 to 1 mg l -1 in ponds (Chang et al 2006). Though trichlorfon half-life time in the water was short (57 h; Lopes et al 2006), excessive amounts are often applied in fish and agriculture farm management. Residues of trichlorfon in the water have caused problems in non-targeted species, such as fish and shrimps (Yeha et al 2005;Guimara et al 2006).…”

Section: Introductionmentioning

confidence: 99%

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Effect of trichlorfon on oxidative stress and hepatocyte apoptosis of Carassius auratus gibelio in vivo

Liu

et al. 2011

Fish Physiol Biochem

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To investigate the effect of trichlorfon on oxidative stress and hepatocyte apoptosis of Carassius auratus gibelio in vivo, the fish were exposed to 0, 0.5, 1.0, 2.0 and 4.0 mg l(-1) trichlorfon concentrations for 30 days. At the end of the experiment, plasm antioxidant activities, hepatic total nitric oxide synthase (T-NOS) activity, xanthine oxidase (XOD) activity and hepatocyte apoptosis rate were analyzed. The results showed hepatic T-NOS activities, XOD activities and hepatocyte apoptosis rate were increased with trichlorfon concentration increase. The hepatic MDA content was increased significantly at 4.0 mg l(-1) trichlorfon treatment. The disturbance of antioxidative balance was observed based on the monitoring of the plasm superoxide dismutase (SOD), catalase (CAT) activities and vitamin E levels. The current study revealed that trichlorfon influenced fish plasma antioxidative status, caused lipid peroxidation (LPO) then resulted in hepatocytes apoptosis.

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Section: Experimental Designmentioning

confidence: 99%

Section: Introductionmentioning

confidence: 99%

Effect of trichlorfon on oxidative stress and hepatocyte apoptosis of Carassius auratus gibelio in vivo

Liu

et al. 2011

Fish Physiol Biochem

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show abstract

“…Bioconcentration of trichlorfon was assessed in pacu (Lopes et al, 2006), but there are no studies on the effects of this organic pesticide in this fish. The present study determined toxic concentrations of trichlorfon to pacu through the LC 50 ⁄ 96 h and histopathological alterations to the gills, liver and kidney that are provoked by this organophosphate compound.…”

Section: Introductionmentioning

confidence: 99%

Histopathological changes in the gill, liver and kidney of pacu (Piaractus mesopotamicus, Holmberg, 1887) exposed to various concentrations of trichlorfon

Mataqueiro

Nakaghi

Souza

et al. 2009

Journal of Applied Ichthyology

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“…Lopes et al, (2006) establish that for the fish "pacu" Piractus mesopotamicus exposed in culture to trichlorfon, the compound showed low half-life, medium half-life and a low concentration in tissues. They also mention that 50 days are required for the elimination of the compound in the fish.…”

Section: Conclusionesmentioning

confidence: 78%