Bilateral coracoclavicular joint

Nehme, I.; Tricoire, J; Colombier, Daniel; Giordano, G.; Feghaly, M. El; Puget, J.

doi:10.1007/bf01681656

Cited by 3 publications

(5 citation statements)

References 4 publications

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“…Most sources claim the condition is “rare” (Cheung et al, 2006; Faraj, 2003; Gibbs et al, 2014; Gupta et al, 2015; Lanotte & Van Den Broeck, 2015; Moore & Renner, 1957; Nehme et al, 1998; Olotu et al, 2008; Singh et al, 2009; Singh et al, 2011; Wertheimer, 1948). However, Mann et al (2016) describe it as “uncommon,” Lewis (1959) regards it as “common,” and De Haas et al (1965) state that it is “far from being rare”.…”

Section: The Coracoclavicular Jointmentioning

confidence: 99%

“…Radiographs are readily available and provide an opportunity to create large sample sizes, however, there are a couple of caveats worth noting. Nehme et al (1998) point out that during the process of taking a chest X‐ray, axial rotation of the clavicle can exaggerate the size of the normal conoid tubercle and this could be mistaken as a CCJ. Also, if the outgrowth is missing but there is still a joint capsule and/or articular cartilage present, the joint would probably not be observed and thus not counted as a CCJ (Kaur & Jit, 1991; Lewis, 1959).…”

Section: The Coracoclavicular Jointmentioning

confidence: 99%

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A review of the coracoclavicular joint: Description, etiology, and clinical significance

Williamson

2023

Clinical Anatomy

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The coracoclavicular joint (CCJ) is a synovial joint that forms between the conoid tubercle of the clavicle and the coracoid process of the scapula in approximately 2.5% of the population. The number of bilateral to unilateral cases is almost equal.The number of right-sided and left-sided cases is also almost equal. It is found in both males and females but most often in male adults. Very few cases have been identified in juveniles. Found in populations all over the world, the highest frequencies of CCJ are in Asia. The etiology is unknown but it is most likely caused by metaplastic change of the trapezoid and surrounding tissue due to compression and friction of the coracoacromial ligament between the clavicle and coracoid process. Typically asymptomatic, but if so, the most common complaint is anterior should pain exacerbated by extreme abduction. Successful treatment includes steroid injection and surgical excision.

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Section: The Coracoclavicular Jointmentioning

confidence: 99%

Section: The Coracoclavicular Jointmentioning

confidence: 99%

A review of the coracoclavicular joint: Description, etiology, and clinical significance

Williamson

2023

Clinical Anatomy

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“…From a biomechanical point of view, the coracoclavicular ligaments are very important in maintaining the vertical and horizontal stability of the acromioclavicular joint. In addition, the little known coracoclavicular joint, which is the cause of shoulder pain and upper limb paresthesia, is an abnormal joint formed between the horizontal part of the coracoid process of the scapula and the lower surface of the clavicle [2,13,22,28,29].…”

Section: Introductionmentioning

confidence: 99%

The morphometric features, variability and clinical significance of the coracoclavicular tuberosity (trapezoid line and conoid tubercle)

Akbulut,

Karakaş,

Yarar

et al. 2024

Preprint

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Purpose: The coracoclavicular ligament attaches to the coracoclavicular tuberosity, and consists of the conoid and trapezoid ligaments. It plays an important role in the stability of the acromioclavicular joint. In addition, it is clinically important that the little-known coracoclavicular joint causes shoulder pain and upper extremity paresthesias. The aim of this study was to create an osteologic map of the morphologic projection of the coracoclavicular ligament or joint components on the clavicle. Methods: In this study, 93 dry clavicles without age and sex records were used. The attachment sites of the trapezoid and conoid ligaments, which form the coracoclavicular ligament, were examined structurally and formally. Results: The attachment sites of the trapezoid ligament were found to be triangular in 9 clavicles, ellipsoidal in 18 clavicles and trapezoid in 62 clavicles. Anatomical structure of the attachment sites of the trapezoid ligament; 57 were tuberosities, 12 were tubercles, 11 were line and 9 were fovea. The attachments of the conoid ligament on the clavicle were found to be 23 weak, 37 moderate and 29 strong. Anatomical structure of the attachment sites of the conoid ligament; 50 were tubercle, 20 were tuberosity, 8 were massa, 6 were crista, 3 were impressio and 2 were spina. The attachments of the conoid ligament on the clavicle were found to be 20 weak, 50 moderate and 19 strong. The incidence of coracocalavicular joint was 6% in this study. Conclusion: In conclusion, this study provides an osteologic map of the morphologic projections of the components of the coracoclavicular ligament or joint in the clavicle. It was thought that the results of this study could shed light on surgeons in orthopedic surgery and clinicians in the field of physical therapy.

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“…Indeed, recent clinical studies have shown that systemic bisphosphonate treatment following a prosthesis implantation reduced peri-implant bone loss only 3 months after the treatment [4,5]. Moreover, proximal femur, the region where most of the bone resorption occurs, is reached with difficulty by bisphosphonate systemically injected as shown by the lower decrease of femur fracture prevention compared to vertebra fracture prevention in a phase III clinical studies for osteoporosis treatment with systemic bisphosphonate treatment [10].…”

Section: Is There a Clinical Need For Local Delivery Of Drug Around Omentioning

confidence: 99%

“…Decreasing the catabolic bone activity would then be a good strategy to avoid peri-implant bone loss. The drug of choice so far is of bisphosphonate type and several clinical trials have been performed and effectively show a reduction of peri-implant bone loss in treated groups [4][5][6]. Systemic injections of a drug may then be potentially interesting to control the bone remodeling around orthopedic implant, however this mode of delivery may not be optimal.…”

Section: Is There a Clinical Need For Local Delivery Of Drug Around Omentioning

confidence: 99%

Orthopedic Implant Used as Drug Delivery System: Clinical Situation and State of the Research

Pioletti¹,

Gauthier²,

Stadelmann³

et al. 2008

CDD

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Abstract:A partial review is proposed on the existing literature for the research performed in orthopedic implant used as drug delivery system. In the first part, an evaluation is given on the clinical need to deliver a drug in the surrounding of an implant. Secondly, a review of the clinical situation is developed for implants already used as drug delivery system. Experimental works performed for local delivery are reported. In particular, a description is given on the in vitro and in vivo studies where the implant is coated with different proteins or drugs. Finally, a conclusion is proposed on the next step in the development of orthopedic implant as drug delivery system mentioning also the industrial situation.Keywords: Orthopedic implant, local delivery, drug, bone remodelling. IS THERE A CLINICAL NEED FOR LOCAL DELIVERY OF DRUG AROUND ORTHOPEDIC IMPLANT?Orthopedic practice recently enters a new area by considering use of drugs to enhance the fixation of implants. The rational behind this approach is to keep the bone around the implant as the trend observed clinically is a peri-implant bone resorption for hip or knee implants. Studies over the past 2 decades have strongly implicated osteoclasts as the major cause of the bone lysis leading to implant failure [1,2]. Different activation pathways of osteoclasts have been identified and in particular the RANKL/OPG/RANK pathway has been given much interest [3]. Decreasing the catabolic bone activity would then be a good strategy to avoid peri-implant bone loss. The drug of choice so far is of bisphosphonate type and several clinical trials have been performed and effectively show a reduction of peri-implant bone loss in treated groups [4][5][6]. Systemic injections of a drug may then be potentially interesting to control the bone remodeling around orthopedic implant, however this mode of delivery may not be optimal.The failure of an orthopedic implant is strongly correlated to its bone fixation [7]. When an implant is used without cement, stability immediately after the surgery must be obtained, a process called primary fixation, followed by a long-term fixation, a process called secondary fixation. A race on fixation quality is then engaged with the concept that the faster the secondary implant fixation is obtained, the better will be the implant outcome. The primary fixation being insured by the press-fit technique, the drug should then target the secondary fixation with the goal of reducing the bone loss. We should not be mislead by the term secondary fixation which is indeed already happening during the first months following the surgery. This has been shown in a clinical study where up to 14% bone loss arose during the first three months after total hip arthroplasties [8]. In parallel, rapid early migrations have been detected by roentgen stereophotogrammetry in many asymptotic hips, often as early as four months postoperatively [9]. The migrations have been found to predict an increased risk of clinical loosening.Systemic delivery of bisphosphonate by sever...

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Bilateral coracoclavicular joint

Cited by 3 publications

References 4 publications

A review of the coracoclavicular joint: Description, etiology, and clinical significance

A review of the coracoclavicular joint: Description, etiology, and clinical significance

The morphometric features, variability and clinical significance of the coracoclavicular tuberosity (trapezoid line and conoid tubercle)

Orthopedic Implant Used as Drug Delivery System: Clinical Situation and State of the Research

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