Automatic repetitive decompression of the saccule in endolymphatic hydrops (tack operation): Preliminary report

Cody, D. Thane R.; Simonton, Kinsey M.; Hallberg, O. Erik

doi:10.1288/00005537-196708000-00017

Cited by 50 publications

(20 citation statements)

References 24 publications

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“…Two mechanisms are generally acknowledged to be involved in a Tullio phenomenon in humans: more than one mobile window or a fistula opens into the vestibular labyrinth [41,42] and there is a pathological contiguity of the tympano-ossicular chain and the membranous labyrinth, for example, if the stapes is in contact with the saccule because of endolymphatic hydrops [42][43][44].…”

Section: Clinical Types Of Tullio Phenomenamentioning

confidence: 99%

Otolithic Vertigo

Brandt¹

2000

Otolith Function and Disorders

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There are several reasons why most vestibular syndromes involve both semicircular canal function and otolithic function:(1) The different receptors for perception of angular and linear accelerations are housed in a common labyrinth.(2) Their peripheral (eighth nerve) and central (e.g. medial longitudinal fascicle) pathways take the same course.(3) Otolith and semicircular canal input converge at all central vestibular levels, from the vestibular nuclei to the vestibular cortex.Thus, most vestibular syndromes are mixed as regards otolithic and canal function. A peripheral prototype of such a mixed syndrome is vestibular neuritis. It is caused by inflammation of the superior division of the vestibular nerve that subserves the horizontal and the anterior semicircular canals and the maculae of the utricle and the anterosuperior part of the saccule. A central prototype is Wallenberg's syndrome. In this disorder the medial and superior vestibular nuclei are involved where otolith and canal input converge. Wallenberg's syndrome typically causes ocular and body lateropulsion and torsional spontaneous nystagmus.Nevertheless, with caloric irrigation of the external acoustic meatus it is possible to selectively stimulate single canals. The prototype of a semicircular canal disease is benign paroxysmal positioning vertigo of the posterior or horizontal canal. Typical signs and symptoms of semicircular canal vertigo are:(a) Rotational vertigo and deviation of perceived straight-ahead. (b) Spontaneous vestibular nystagmus with oscillopsia. (c) Postural imbalance with Romberg fall and pastpointing. (d) Nausea and vomiting if severe.

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Section: Clinical Types Of Tullio Phenomenamentioning

confidence: 99%

Otolithic Vertigo

Brandt¹

2000

Otolith Function and Disorders

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“…This operation involved implanting a tack prosthesis into the vestibule that would decompress the saccule during hydropic episodes. 36 In this surgery, a 1.0-to 1.9-mm stainless steel tack was introduced via a transcanal stapedectomy approach using a magnetic probe through the anteroinferior aspect of the footplate (Figure 7). 38 In reviewing the first 290 cases, Cody reported that vertigo was satisfactorily controlled in 79% of patients and hearing was improved or maintained in greater than 60%.…”

Section: Ménière Diseasementioning

confidence: 99%

The History of Otologic Surgery at Mayo Clinic, 1883 to Present

Carlson

2019

Mayo Clinic Proceedings

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“…In the 1960ies, a procedure was described that should lead to relief of the ELH by a perilymphatic/endolymphatic shunt [ 108 ], [ 109 ], [ 110 ], [ 111 ]. It was applied in patients with therapy-resistant vertigo after failure of alternative therapeutic options.…”

Section: Hydropic Inner Ear Diseases – Menière’s Disease and Menièrmentioning

confidence: 99%