Atrial natriuretic factor and sympathetic nervous output in response to volume shifts in the immature canine circulation

Ross, Robert D.; Womack, Sophie J; Gunther, Stephen; Karpawich, Peter P.; Justice, Cheryl D.; Pinsky, William W.

doi:10.1016/0196-9781(91)90173-m

Cited by 3 publications

(1 citation statement)

References 16 publications

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“…These inflammatory cells include macrophages, T-lymphocytes (Bjorkerud and Bjorkerud 1996), and mast cells (Kovanen et al 1995). Several growth factors have been identified in atherosclerotic plaques (Ross et al 1991) and restenotic lesions (Cercek et al 1991), and they too induce the migration and proliferation of VSMCs. Curproteins from migrating into the vessel wall under normal conditions (Sumpio et al 2002).…”

Section: Introductionmentioning

confidence: 98%

Cells, cytokines and cellular immunity in the pathogenesis of fibroproliferative vasculopathies

Mitra¹,

Core²,

Agrawal³

2005

Can. J. Physiol. Pharmacol.

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Atherosclerosis and restenosis are the result of vascular injury followed by an inflammatory and fibroproliferative response that involves a large number of growth factors, cytokines, and cellular elements. Platelet activation and leukocyte recruitment into the arterial intima play a crucial role, initiating a whole spectrum of reactions leading to vascular smooth muscle cell hyperplasia and intimal migration. The roles of macrophages and lymphocytes and mast cells as mediators of inflammation and immune response is discussed, as are the roles of growth factors and cytokines. New light on the 'old' problems will help us to devise newer and better therapeutic strategies to combat these clinical entities.

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Section: Introductionmentioning

confidence: 98%

Cells, cytokines and cellular immunity in the pathogenesis of fibroproliferative vasculopathies

Mitra¹,

Core²,

Agrawal³

2005

Can. J. Physiol. Pharmacol.

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Effect of oral captopril on premature ventricular complexes in patients with preserved left ventricular systolic function: A randomized clinical trial

Steinman

Gilley

Gunther

et al. 1993

Clinical Cardiology

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Summary: Some studies have suggested that angiotensinconverting enzyme (ACE) inhibition in patients with heart failure is associated with a decrease in frequency of spontaneous premature ventricular complexes (PVCs). It is not clear whether such a finding represents a primary effect of ACE inhibition or, instead, a secondary result of treatment of heart failure. For a primary drug effect to be present, PVC suppression during ACE inhibition should also occur in patients with preserved left ventricular systolic function. We therefore undertook a randomized double-blind placebo-controlled crossover clinical trial to assess the effect of oral captopril(50 mg. bid.) on ventricular arrhythmia frequency in 11 patients with > 30 PVCs/h (during a 48-h ambulatory recording) and a left ventricular ejection fraction of 2 45% (measured by radionuclide multigated acquisition scan). Pharmacologic activity of the administered drug was evidenced by an increase in median plasma renin activity to 2.4 ng/AymVh from a value of 1.1 ng/AI/nVh during placebo (p = 0.001) and an 8.5 f 10.3 drop in mean diastolic blood pressure during captopril versus placebo (p < 0.03).

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Increased atrial natriuretic factor response to exercise after coarctation repair

Ross

Clapp

Gunther

et al. 1992

The American Journal of Cardiology

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Atrial natriuretic factor and sympathetic nervous output in response to volume shifts in the immature canine circulation

Cited by 3 publications

References 16 publications

Cells, cytokines and cellular immunity in the pathogenesis of fibroproliferative vasculopathies

Cells, cytokines and cellular immunity in the pathogenesis of fibroproliferative vasculopathies

Effect of oral captopril on premature ventricular complexes in patients with preserved left ventricular systolic function: A randomized clinical trial

Increased atrial natriuretic factor response to exercise after coarctation repair

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